Makoto Kanzaki

Professor

Tohoku University

Japan

Research Interests

Explore related searches

Professors in Japan

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

About

Prof. Makoto Kanzaki is a professor at Tohoku University in Japan. His research focuses on areas such as glucose metabolism, muscle function, and the antibacterial properties of materials. Recent articles include explorations of insulin-responsive intracellular GLUT4 trafficking and mitochondrial dysfunction in myositis. He also investigates innovative imaging techniques for understanding muscle activity and cell structures.

Articles (11)

Year:

2025

Year:

2023

Year:

2022

Year:

2022

Year:

2021

Year:

2021

Year:

2020

Year:

2020

Year:

2020

Year:

Tohoku University

JAPAN

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Makoto Kanzaki

Research Interests

Explore related searches

Contact this professor

About

Articles (11)

CEFIP deficiency in mice enhances glucose tolerance despite compromised muscle function

Effects of CX3CR1 and CXCR2 antagonists on running-dependent intramuscular neutrophil recruitments and myokine upregulation

A comparative study of the antibacterial properties of copper-based transparent oxides at the solid–liquid interface

Three live-imaging techniques for comprehensively understanding the initial trigger for insulin-responsive intracellular GLUT4 trafficking

Blood glucose‐lowering effect of water and ethanolic extracts of Gynura procumbens (Lour.) Merr

Imaging of muscle activity‐induced morphometric changes in fibril network of myofascia by two‐photon microscopy

Characterization of middle-molecule introduction into cells using mm-scale discharge in saline

Continuous release of O2−/ONOO− in plasma‐exposed HEPES‐buffered saline promotes TRP channel‐mediated uptake of a large cation

TRPA1 and TRPV1 channels participate in atmospheric-pressure plasma-induced [Ca2+]i response

Mitochondrial dysfunction underlying sporadic inclusion body myositis is ameliorated by the mitochondrial homing drug MA-5.

Development of optical resolution photoacoustic microscopy with sub-micron lateral resolution for visualization of cells and their structures

Collaborators (6)

Yasutoshi Akiyama

Shota Sasaki

Keisuke Takashima

Toshiro Kaneko

Ken-ichiro Hayashi

Masahiro Tsuchiya

Make your next move.