Malgorzata Kajta

Professor

Institute of Pharmacology

Poland

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About

Professor Malgorzata Kajta is a faculty member at the Maj Institute of Pharmacology in Poland. Her research focuses on neuroprotection, epigenetic mechanisms, and the effects of environmental factors on neurodegeneration. Recent articles authored by Professor Kajta explore topics such as the roles of PPARγ in inflammation, the impact of prenatal exposure to chemicals on brain development, and potential treatment strategies for Alzheimer's disease. She is recognized for her contributions to understanding mitochondrial status and gene regulation in neurobiological contexts.

Articles (11)

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2024

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2024

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2023

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2021

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2021

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2021

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2021

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2020

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2020

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2020

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2020

Collaborators (4)

Elzbieta Salinska

Head of Department/professor

Mossakowski Medical Research Centre

POLAND

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Julita Wesołowska

Research and teaching assistant

Jagiellonian University

POLAND

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Anna K. Wójtowicz

professor

University of Agriculture

POLAND

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Bernadeta Angelika Pietrzak-Wawrzyńska

Institute of Pharmacology

POLAND

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Malgorzata Kajta

Research Interests

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Articles (11)

Amorfrutin B Compromises Hypoxia/Ischemia-induced Activation of Human Microglia in a PPARγ-dependent Manner: Effects on Inflammation, Proliferation Potential, and Mitochondrial Status

Posttreatment with PaPE-1 Protects from Aβ-Induced Neurodegeneration Through Inhibiting the Expression of Alzheimer’s Disease-Related Genes and Apoptosis Process That Involves Enhanced DNA Methylation of Specific Genes

Amorfrutin B Protects Mouse Brain Neurons from Hypoxia/Ischemia by Inhibiting Apoptosis and Autophagy Processes Through Gene Methylation- and miRNA-Dependent Regulation

Prenatal Exposure to Triclocarban Impairs ESR1 Signaling and Disrupts Epigenetic Status in Sex-Specific Ways as Well as Dysregulates the Expression of Neurogenesis- and Neurotransmitter-Related Genes in the Postnatal Mouse Brain

Posttreatment Strategy Against Hypoxia and Ischemia Based on Selective Targeting of Nonnuclear Estrogen Receptors with PaPE-1

Post-Treatment with Amorfrutin B Evokes PPARγ-Mediated Neuroprotection against Hypoxia and Ischemia

Is the commonly used UV filter benzophenone-3 a risk factor for the nervous system?

Selective Targeting of Non-nuclear Estrogen Receptors with PaPE-1 as a New Treatment Strategy for Alzheimer’s Disease

Autophagy-related neurotoxicity is mediated via AHR and CAR in mouse neurons exposed to DDE

Neuroprotective effect of 3,3’-Diindolylmethane against perinatal asphyxia involves inhibition of the AhR and NMDA signaling and hypermethylation of specific genes

Triclocarban impairs autophagy in neuronal cells and disrupts estrogen receptor signaling via hypermethylation of specific genes

Collaborators (4)

Elzbieta Salinska

Julita Wesołowska

Anna K. Wójtowicz

Bernadeta Angelika Pietrzak-Wawrzyńska

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