Manvendra K. Singh

Assistant Professor

Duke-NUS Medical School

Singapore

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Articles (12)

Year:

2022

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2022

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2022

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2022

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2022

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2021

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2021

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2021

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2021

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2020

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2020

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2020

Collaborators (5)

Brijesh Kumar Singh

Assistant Professor

Duke-NUS Medical School

SINGAPORE

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Hiroki Kurihara

JAPAN

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Derek Hausenloy

University College London

UNITED KINGDOM

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Dinesh Kumar Srinivasan

Associate Professor and Integration Lead Educator

National University of Singapore (NUS)

SINGAPORE

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Fumio Matsuzaki

Specially appointed professor

Kyoto University

JAPAN

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Manvendra K. Singh

Research Interests

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Articles (12)

Emerging roles of the Hippo signaling pathway in modulating immune response and inflammation‐driven tissue repair and remodeling

New Insights into Hippo/YAP Signaling in Fibrotic Diseases

Caffeine prevents restenosis and inhibits vascular smooth muscle cell proliferation through the induction of autophagy

Vitamin B<sub>12</sub> and folate decrease inflammation and fibrosis in NASH by preventing syntaxin 17 homocysteinylation

The E3 ubiquitin ligase WWP2 regulates pro-fibrogenic monocyte infiltration and activity in heart fibrosis.

Cardiac Tissue Factor Regulates Inflammation, Hypertrophy and Heart Failure in Mouse Model of <i>Type 1 Diabetes</i>.

Loss of Yap/taz in cardiac fibroblasts attenuates adverse remodeling and improves cardiac function.

Critical role of the BAF chromatin remodeling complex during murine neural crest development.

Semaphorin3E-PlexinD1 signaling in coronary artery and lymphatic vessel development with clinical implications in myocardial recovery.

Prdm16 Deficiency Leads to Age-Dependent Cardiac Hypertrophy, Adverse Remodeling, Mitochondrial Dysfunction, and Heart Failure

YAP/TAZ deficiency reprograms macrophage phenotype and improves infarct healing and cardiac function after myocardial infarction.

Understanding Inflammatory Responses in the Manifestation of Prothrombotic Phenotypes.

Collaborators (5)

Brijesh Kumar Singh

Hiroki Kurihara

Derek Hausenloy

Dinesh Kumar Srinivasan

Fumio Matsuzaki

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