Marcus Muench

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Adjunct Professor

University of California, San Francisco

United States

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Marcus Muench is an Adjunct Professor at the University of California San Francisco, USA. His research areas include immunology, transfusion medicine, and hematopoietic stem cell biology, as reflected in his recent articles. His work explores topics such as alloimmunity in platelet transfusions, the effects of testosterone on red blood cells, and innovative cryopreservation techniques for human stem cells.

Recent Grants

Grant: Open

Hematopoietic and Immune Development in the Human Chorion

Open Date: 2022-04-11

Close Date: 2027-03-31

Articles (14)

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2020

Collaborators (1)

Kimberly A. Thomas

UNITED STATES

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Marcus Muench

Research Interests

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Recent Grants

Articles (14)

Testosterone supplementation increases red blood cell susceptibility to oxidative stress, decreases membrane deformability, and decreases survival after cold storage and transfusion

A Monocytic Barrier to the Humanization of Immunodeficient Mice

CD203c is expressed by human fetal hepatoblasts and distinguishes subsets of hepatoblastoma.

Polyinosinic: polycytidylic acid induced inflammation enhances while lipopolysaccharide diminishes alloimmunity to platelet transfusion in mice

Enhanced alloresponse to platelet transfusion due to immune dysregulation following ablative chemotherapy in mice

Antibody screening data of human midgestation liver cells with a focus on hematopoietic, liver sinusoidal endothelial, and hepatoblast cell-populations

Frequent detection but lack of infectivity of SARS-CoV-2 RNA in presymptomatic, infected blood donor plasma

Reduced dimethyl sulfoxide concentrations successfully cryopreserve human hematopoietic stem cells with multi-lineage long-term engraftment ability in mice

A bioinspired and chemically defined alternative to dimethyl sulfoxide for the cryopreservation of human hematopoietic stem cells.

Generation of recombinant hyperimmune globulins from diverse B-cell repertoires.

Blood donor obesity is associated with changes in red blood cell metabolism and susceptibility to hemolysis in cold storage and in response to osmotic and oxidative stress

Pathogen-reduced PRP blocks T-cell activation, induces Treg cells, and promotes TGF-β expression by cDCs and monocytes in mice

μ-Lat: A mouse model to evaluate human immunodeficiency virus eradication strategies.

Immunodeficient mice are better for modeling the transfusion of human blood components than wild-type mice

Collaborators (1)

Kimberly A. Thomas

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