Margit Mahlapuu

Professor in Molecular Genetics

Sweden

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Professor Margit Mahlapuu is a faculty member in the Department of Molecular Genetics at the University of Gothenburg, Sweden. Her research focuses on the therapeutic potential of STE20-type kinases in metabolic diseases and liver health, as evidenced by her recent publications on hepatocellular carcinoma, lipid homeostasis, and the role of kinases in metabolic reprogramming. Professor Mahlapuu's work contributes significantly to understanding the molecular mechanisms underlying metabolic disorders and their potential treatments.

Articles (15)

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2025

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2024

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2024

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2024

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2023

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2023

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2023

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2022

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2022

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2022

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2021

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2021

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2021

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2021

Collaborators (8)

Bengt Hallberg

University of Gothenburg

SWEDEN

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Daniel Hutchinson

KTH Royal Institute of Technology

SWEDEN

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derya burcu hazer

Lund University

SWEDEN

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Johan Ruud

Assistant Professor, Senior Lecturer

University of Gothenburg

SWEDEN

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Hanns-Ulrich Marschall

University of Gothenburg

SWEDEN

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Brian Howell

Associate Professor

SUNY Upstate Medical University

UNITED STATES

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Thomas Svensson

Karolinska Institutet

SWEDEN

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Lars B. Dahlin

Linköping University

SWEDEN

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Margit Mahlapuu

Research Interests

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Articles (15)

Therapeutic Potential of STE20-Type Kinase STK25 Inhibition for the Prevention and Treatment of Metabolically Induced Hepatocellular Carcinoma.

PXL01 alters macrophage response with no effect on axonal outgrowth or Schwann cell response after nerve repair in rats

Genetic Ablation of STE20-Type Kinase MST4 Does Not Alleviate Diet-Induced MASLD Susceptibility in Mice

GCKIII kinases control hepatocellular lipid homeostasis via shared mode of action.

Dendritic Nanogels Directed Dual‐Encapsulation Topical Delivery System of Antimicrobial Peptides Targeting Skin Infections

STE20‐type kinases MST3 and MST4 promote the progression of hepatocellular carcinoma: Evidence from human cell culture and expression profiling of liver biopsies

Silencing of STE20-type kinase TAOK1 confers protection against hepatocellular lipotoxicity through metabolic rewiring.

Knockout of STE20-type kinase TAOK3 does not attenuate diet-induced NAFLD development in mice.

Inhibition of MAP4K4 signaling initiates metabolic reprogramming to protect hepatocytes from lipotoxic damage.

GCKIII kinases in lipotoxicity: Roles in NAFLD and beyond.

Silencing of STE20-type kinase STK25 in human aortic endothelial and smooth muscle cells is atheroprotective.

STE20-Type Kinase TAOK3 Regulates Hepatic Lipid Partitioning

Evaluation of LL‐37 in healing of hard‐to‐heal venous leg ulcers: A multicentric prospective randomized placebo‐controlled clinical trial

Antagonizing STK25 Signaling Suppresses the Development of Hepatocellular Carcinoma Through Targeting Metabolic, Inflammatory, and Pro-Oncogenic Pathways.

Silencing of STE20-type kinase MST3 in mice with antisense oligonucleotide treatment ameliorates diet-induced nonalcoholic fatty liver disease.

Collaborators (8)

Bengt Hallberg

Daniel Hutchinson

derya burcu hazer

Johan Ruud

Hanns-Ulrich Marschall

Brian Howell

Thomas Svensson

Lars B. Dahlin

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