Marie Lagerquist

Associate Professor

University of Gothenburg

Sweden

Research Interests

Explore related searches

Professors in Sweden

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

About

Marie Lagerquist is an Associate Professor at the University of Gothenburg, Sweden. Her research areas focus on the mechanisms of hormone signaling, bone health, and the effects of various dietary components on male reproductive physiology, as evidenced by her recent publications. Notable works include studies on the estrogen signaling's impact on skeletal response and the role of local antibody distribution in bone resorption.

Articles (15)

Year:

2024

Year:

2024

Year:

2023

Year:

2023

Year:

2023

Year:

2022

Year:

2022

Year:

2022

Year:

2022

Year:

2022

Year:

2022

Year:

2022

Year:

2021

Year:

2021

Year:

2020

Collaborators (9)

Klara Sjögren

University of Gothenburg

SWEDEN

View details

Hannah Colldén

University of Gothenburg

SWEDEN

View details

Cecilia Engdahl

SWEDEN

View details

Matti Poutanen

professor

FINLAND

View details

Ulf Lerner

Professor

University of Gothenburg

SWEDEN

View details

U Islander

University of Gothenburg

SWEDEN

View details

Sara Windahl

Assistant Senior Lecturer

Karolinska Institutet-Huddinge Campus

SWEDEN

View details

Petra Henning

University of Gothenburg

SWEDEN

View details

Alexandra Stubelius

Associate Professor at Chalmers University of Technology

Chalmers University of Technology

SWEDEN

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Marie Lagerquist

Research Interests

Explore related searches

Contact this professor

About

Articles (15)

Consistent and effective method to define the mouse estrous cycle stage by deep learning based model.

Essential role of local antibody distribution in mediating bone-resorbing effects.

Membrane estrogen receptor α signaling modulates the sensitivity to estradiol treatment in a dose- and tissue- dependent manner.

Dietary Progesterone Contributes to Intratissue Levels of Progesterone in Male Mice.

B4GALNT3 regulates glycosylation of sclerostin and bone mass.

Phosphorylation of S122 in ERα is important for the skeletal response to estrogen treatment in male mice

Dehydroepiandrosterone Supplementation Results in Varying Tissue-specific Levels of Dihydrotestosterone in Male Mice

Physiological levels of estradiol limit murine osteoarthritis progression.

Reduction of Mature B Cells and Immunoglobulins Results in Increased Trabecular Bone.

ERα Signaling in a Subset of CXCL12-Abundant Reticular Cells Regulates Trabecular Bone in Mice.

A tissue-specific role of membrane-initiated ERα signaling for the effects of SERMs.

A tissue-selective estrogen complex as treatment of osteoporosis in experimental lupus.

Pulsed administration for physiological estrogen replacement in mice.

Acute fat loss does not affect bone mass.

Immunoglobulin G complexes without sialic acids enhance osteoclastogenesis but do not affect arthritis-mediated bone loss

Collaborators (9)

Klara Sjögren

Hannah Colldén

Cecilia Engdahl

Matti Poutanen

Ulf Lerner

U Islander

Sara Windahl

Petra Henning

Alexandra Stubelius

Make your next move.