Marin Barisic

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Associate Professor

University of Copenhagen

Denmark

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About

Marin Barisic is an Associate Professor at the University of Copenhagen in Denmark. His research focuses on cell division mechanisms, particularly concerning microtubule behavior, chromosome dynamics, and the role of the cytoskeleton in directed cell migration. Recent articles authored by Professor Barisic delve into topics such as anaphase bridge signaling and the activation of CENP-E during mitosis.

Recent Grants

Grant: Close

Tubulin post-translational modifications in cancer - the impact on aneuploidy and metastasis

Open Date: 2017-01-01

Close Date: 2019-12-01

Grant: Close

Deciphering the 'tubulin code' in mitosis and cancer

Open Date: 2016-09-01

Close Date: 2021-08-01

Articles (13)

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2025

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2025

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2024

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2023

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2023

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2023

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2022

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2022

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2022

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2021

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2021

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2021

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2020

Collaborators (4)

Elisa Vitiello

University of Oxford

UNITED KINGDOM

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Andrew Goryachev

University of Edinburgh

UNITED KINGDOM

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Arockia Jeyaprakash Arulanandam

Professor

Ludwig-Maximilians-Universität München

GERMANY

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Jakob Nilsson

Professor

University of Copenhagen

DENMARK

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Marin Barisic

Research Interests

Contact this professor

About

Recent Grants

Articles (13)

Mechanistic insight into anaphase bridge signaling to the abscission checkpoint

How does the tubulin code facilitate directed cell migration?

A conserved CENP-E region mediates BubR1-independent recruitment to the outer corona at mitotic onset.

SiR-DNA/SiR–Hoechst-induced chromosome entanglement generates severe anaphase bridges and DNA damage

CENP-E activation by Aurora A and B controls kinetochore fibrous corona disassembly

Microtubule detyrosination drives symmetry breaking to polarize cells for directed cell migration

Chemogenetic profiling reveals PP2A-independent cytotoxicity of proposed PP2A activators iHAP1 and DT-061.

Kinetochore- and chromosome-driven transition of microtubules into bundles promotes spindle assembly.

Posttranslational modification of microtubules by the MATCAP detyrosinase.

TH588 and Low-Dose Nocodazole Impair Chromosome Congression by Suppressing Microtubule Turnover within the Mitotic Spindle

Mitotic poleward flux: Finding balance between microtubule dynamics and sliding.

The metaphase spindle at steady state - Mechanism and functions of microtubule poleward flux.

Spatially and temporally defined lysosomal leakage facilitates mitotic chromosome segregation

Collaborators (4)

Elisa Vitiello

Andrew Goryachev

Arockia Jeyaprakash Arulanandam

Jakob Nilsson

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