Mark Haid

Head Of Lipidomics Platform

Germany

Research Interests

Explore related searches

Professors in Germany

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

About

Mark Haid is the Head of the Lipidomics Platform at Helmholtz Zentrum München German Research Center for Environmental Health in Germany. His research focuses on lipid metabolism and its implications in cardiovascular diseases, diabetes, and obesity, as evident from his recent publications. He has contributed to studies involving metabolomic profiling, molecular phenotype analysis, and the impact of maternal health on metabolic outcomes in neonatal offspring.

Articles (15)

Year:

2025

Year:

2024

Year:

2024

Year:

2024

Year:

2024

Year:

2023

Year:

2023

Year:

2023

Year:

2023

Year:

2022

Year:

2022

Year:

2021

Year:

2021

Year:

2020

Year:

2020

Collaborators (33)

Helle Krogh Pedersen

Københavns Universitet

DENMARK

Mark Haid

Research Interests

Explore related searches

Contact this professor

About

Articles (15)

An atypical atherogenic chemokine that promotes advanced atherosclerosis and hepatic lipogenesis

The Association of Cardiometabolic, Diet and Lifestyle Parameters With Plasma Glucagon-like Peptide-1

Concordant inter-laboratory derived concentrations of ceramides in human plasma reference materials via authentic standards

Role of human plasma metabolites in prediabetes and type 2 diabetes from the IMI-DIRECT study.

Endothelial epoxyeicosatrienoic acid release is intact in aldosterone excess

Discovery of drug–omics associations in type 2 diabetes with generative deep-learning models

Genetic analysis of blood molecular phenotypes reveals common properties in the regulatory networks affecting complex traits

Maternal hyperglycemia induces alterations in hepatic amino acid, glucose and lipid metabolism of neonatal offspring: Multi-omics insights from a diabetic pig model.

The aryl hydrocarbon receptor regulates lipid mediator production in alveolar macrophages

Four groups of type 2 diabetes contribute to the etiological and clinical heterogeneity in newly diagnosed individuals: An IMI DIRECT study

Trip13 Depletion in Liver Cancer Induces a Lipogenic Response Contributing to Plin2-Dependent Mitotic Cell Death.

Cross-Laboratory Standardization of Preclinical Lipidomics Using Differential Mobility Spectrometry and Multiple Reaction Monitoring.

Fibroblast growth factor induced Ucp1 expression in preadipocytes requires PGE2 biosynthesis and glycolytic flux

Dietary metabolite profiling brings new insight into the relationship between nutrition and metabolic risk

High levels of modified ceramides are a defining feature of murine and human cancer cachexia

Collaborators (33)

Helle Krogh Pedersen

Konstantinos Tsirigos

Anna Nicolaou

Jakob Nissen

Mathias Uhlen

Jochen M Schwenk

Andreas J. Hülsmeier

Tue Hansen

Colin Palmer

Ewan Pearson

Cecilia Engel Thomas

Alexander Bartelt

Oluf Pedersen

Stephanie Roessler

Nisha Shah

Yassene Mohammed

Henry Webel

Holger Schneider

Angus Jones

Joanna Darck Carola Correia Lima

Torben Hansen

Cristina Coman

Federico De Masi

Tarja Kokkola

Anders Gorm Pedersen

Henrik Vestergaard

Britta Brügger

Yong Fan

Agnete Troen Lundgaard

Roldan de Guia

Harriet Teare

Anna Jonsson

Mauricio Berriel Diaz

Make your next move.