Martina Muckenthaler

Prof. Dr. phil. nat.

University Medical Center

Germany

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Prof. Dr. phil. nat. Martina Muckenthaler is a faculty member at University Medical Center in Germany, where she focuses her research on innate immunity, iron homeostasis, and inflammatory responses. Her recent work includes exploring the role of RBM39 in the interferon response and the impact of hepatocyte Toll-like receptors on hepcidin during pathogen interactions. Additionally, she investigates the effects of macrophage depletion on iron overload-related conditions and the regulatory mechanisms of iron biomarkers in disease.

Articles (20)

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2025

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2023

Collaborators (12)

Dania Fischer

Heidelberg University

GERMANY

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Gianluca Brugnara

University Hospital Heidelberg

GERMANY

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Ursula Klingmüller

Head

Deutsches Krebsforschungszentrum

GERMANY

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Sandro Altamura

Heidelberg University

GERMANY

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Michael Forster

Kiel University

GERMANY

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Silvia Colucci

Heidelberg University

GERMANY

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Martin Burtscher

Professor/MD,PhD

University of Innsbruck

AUSTRIA

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Christina Mertens

University Hospital Heidelberg

GERMANY

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Jose Manuel Fernández-Real

Full Professor

Universitat de Girona

SPAIN

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Christopher Neufeldt

Assistant Professor

Emory University School of Medicine

UNITED STATES

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Irene Motta

University of Milan

ITALY

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Walee Chamulitrat

University Hospital Heidelberg

GERMANY

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Martina Muckenthaler

Research Interests

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Articles (20)

RBM39 shapes innate immunity by controlling the expression of key factors of the interferon response.

Hepatocyte Toll‐like receptors contribute to the hepcidin inflammatory response to pathogens and pathogen‐derived ligands

Inflammation-driven NF-κB signaling represses ferroportin transcription in macrophages via HDAC1 and HDAC3.

Iron biomarkers predict peripheral artery disease in females.

Dynamic YAP expression in the non-parenchymal liver cell compartment controls heterologous cell communication

Depletion of macrophages and osteoclast precursors mitigates iron overload-mediated bone loss.

E3 ubiquitin ligases SMURF1 and HECW1 regulate hepcidin-induced degradation of ferroportin in HeLa cells.

Author Correction: Mechanisms controlling cellular and systemic iron homeostasis

Iron scavenging and myeloid cell polarization.

Recommendations for diagnosis, treatment, and prevention of iron deficiency and iron deficiency anemia

Association Between Ferritin Levels and Altitude-Dependent Cardiorespiratory Fitness in Mountain Guides

Myeloid-specific fatty acid transport protein 4 deficiency induces a sex-dimorphic susceptibility for nonalcoholic steatohepatitis in mice fed a high-fat, high-cholesterol diet

Consensus Statement on the definition and classification of metabolic hyperferritinaemia

FKBP12 inhibits hepcidin expression by modulating BMP receptors interaction and ligand responsiveness in hepatocytes

The Increase in Hemoglobin Concentration With Altitude Differs Between World Regions and Is Less in Children Than in Adults.

Is iron deficiency caused by <i>BMPR2</i> mutations or dysfunction in pulmonary arterial hypertension patients?

Low level of antioxidant capacity biomarkers but not target overexpression predicts vulnerability to ROS-inducing drugs.

Interpreting Iron Homeostasis in Congenital and Acquired Disorders.

T cell-independent eradication of experimental glioma by intravenous TLR7/8-agonist-loaded nanoparticles.

Chemotherapy-related hyperbilirubinemia in pediatric acute lymphoblastic leukemia: a genome-wide association study from the AIEOP-BFM ALL study group.

Collaborators (12)

Dania Fischer

Gianluca Brugnara

Ursula Klingmüller

Sandro Altamura

Michael Forster

Silvia Colucci

Martin Burtscher

Christina Mertens

Jose Manuel Fernández-Real

Christopher Neufeldt

Irene Motta

Walee Chamulitrat

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