Masaki Arioka

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Lecturer/Associate Professor

University of Occupational and Environmental Health Japan

Japan

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About

Masaki Arioka is an Associate Professor at the University of Occupational and Environmental Health Japan. His research primarily focuses on the mechanisms of cardiac remodeling, the development of anti-fibrotic agents, and cancer biology, particularly in the context of breast cancer and triple-negative breast cancer. Recent publications include studies on the effects of small molecules in inflammatory responses and the role of signaling pathways in cancer metastasis. He contributes significantly to the understanding of therapeutic strategies in both cardiovascular and cancer-related fields.

Recent Grants

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Development of antitumor drug through M2-type tumor-related macrophage (TAM) regulation by differentiation-inducing factor-1

Open Date: 2021-01-01

Close Date: 2022-01-01

Grant: Close

Elucidation of mechanism of suppressing cancer metastasis by differentiation-inducing factor 1

Open Date: 2020-01-01

Close Date: 2022-01-01

Grant: Close

Recovery of masticatory function in patients with osteoporosis by promoting alveolar bone healing using Liposomal-Wnt3a

Open Date: 2019-01-01

Close Date: 2020-01-01

Grant: Close

Study of anti-tumor activity and anti-metastatic activity of DIF-1 in breast cancer.

Open Date: 2018-01-01

Close Date: 2019-01-01

Grant: Close

The research of anti-tumor effects of differentiation-inducing factor 1 on breast cancer.

Open Date: 2017-01-01

Close Date: 2018-01-01

Articles (10)

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2024

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2023

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2023

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2023

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2022

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2022

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2022

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2021

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2021

Year:

2021

Collaborators (2)

Xue Yuan

Assistant Professor

Indiana University-Purdue University Indianapolis

UNITED STATES

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Jill Helms

Stanford University

UNITED STATES

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Masaki Arioka

Research Interests

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Recent Grants

Articles (10)

2,5-Dimethyl-celecoxib induces early termination of inflammatory responses by transient macrophage accumulation and inhibits the progression of cardiac remodeling in a mouse model of cryoinjury-induced myocardial infarction

Promising small molecule anti-fibrotic agents: Newly developed or repositioned drugs targeting myofibroblast transdifferentiation.

Differentiation-inducing factor 1 activates cofilin through pyridoxal phosphatase and AMP-activated protein kinase, resulting in mitochondrial fission.

DIF-1 exhibits anticancer activity in breast cancer via inhibition of CXCLs/CXCR2 axis-mediated communication between cancer-associated fibroblasts and cancer cells.

Accelerating Socket Repair via WNT3A Curtails Alveolar Ridge Resorption

A WNT protein therapeutic accelerates consolidation of a bone graft substitute in a pre‐clinical sinus augmentation model

Increased risk of metastasis in patients with incidental use of renin-angiotensin system inhibitors: a retrospective analysis for multiple types of cancer based on electronic medical records.

Angiotensin II promotes primary tumor growth and metastasis formation of murine TNBC 4T1 cells through the fibroblasts around cancer cells.

DIF-1 inhibits growth and metastasis of triple-negative breast cancer through AMPK-mediated inhibition of the mTORC1-S6K signaling pathway.

Cardiac and renal protective effects of 2,5-dimethylcelecoxib in angiotensin II and high-salt-induced hypertension model mice.

Collaborators (2)

Xue Yuan

Jill Helms

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