Masami Morimatsu

Associate Prof

Hokkaido University

Japan

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About

Masami Morimatsu is an Associate Professor at Hokkaido University in Japan. His research interests include functional analysis in genetics and molecular biology, with a focus on BRCA2 interactions, mouse behavioral genetics, and cancer biology. Recent articles highlight his work on RAD51 activity, the implications of novel splicing variants, and the effects of specific mutations in cancer-related studies.

Articles (12)

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2024

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2023

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2023

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2022

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2022

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2022

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (3)

Kazuhiko OCHIAI

Associate Professor

Nippon Veterinary and Life Science University

JAPAN

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Teppei NAKAMURA

Associate Professor

Hokkaido University

JAPAN

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Yasunaga Yoshikawa

Kitasato University

JAPAN

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Masami Morimatsu

Research Interests

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Articles (12)

Functional Analysis of Oligoadenylate Synthetase in the Emu (Dromaius novaehollandiae)

A Highly Conserved Region in BRCA2 Suppresses the RAD51-Interaction Activity of BRC Repeats

Verification of quantitative trait loci located on chromosome 4 that affect mouse behaviors by use of congenic and sub-congenic strains generated from DBA/2 and C57BL/6 mice

Canine Mammary Tumor Cell Lines Derived from Metastatic Foci Show Increased RAD51 Expression but Diminished Radioresistance via p21 Inhibition

BRCA2 C-Terminal RAD51-Binding Domain Confers Resistance to DNA-Damaging Agents

In vitro anticancer effects of alpelisib against PIK3CA‑mutated canine hemangiosarcoma cell lines

Reduced translation efficiency due to novel splicing variants in 5′ untranslated region and identification of novel cis-regulatory elements in canine and human BRCA2

The number of glutamines in the N‐terminal of the canine androgen receptor affects signalling intensities

Identification of the core motif of the BRCA2 C-terminal RAD51-binding domain by comparing canine and human BRCA2

The response of adipose tissues to <i>Mycoplasma pulmonis</i> and Sendai virus infection in C57BL/6 and DBA/2 mice

Novel canine isocitrate dehydrogenase 1 mutation Y208C attenuates dimerization ability

The canine RAD51 mutation leads to the attenuation of interaction with PALB2

Collaborators (3)

Kazuhiko OCHIAI

Teppei NAKAMURA

Yasunaga Yoshikawa

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