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Matthew Brewer

Research Assistant Professor

University of Rochester

United States

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About

Matthew Brewer is a Research Assistant Professor at the University of Rochester, United States. His research focuses on the interactions between keratinocytes and pathogens, particularly in the context of skin disorders and viral infections. Recent articles explore the effects of S. aureus on skin barrier function and keratinocyte differentiation, as well as the implications of cytokine signaling in viral susceptibility.

Articles (16)

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2026

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2020

Collaborators (5)

Hector Aguilar-Carreno

Associate Professor of Virology

Cornell University

UNITED STATES

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Benjamin Miller

University of Rochester

UNITED STATES

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Lisa Beck

Deans Professor

University of Rochester

UNITED STATES

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Jos Smits

Staff Scientist

Radboud University Medical Center

NETHERLANDS

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H. Mark Kenney

University of Rochester Medical Center

UNITED STATES

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Matthew Brewer

Research Interests

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About

Articles (16)

Influence of fungi on epithelial homeostasis and role in inflammatory diseases

Bullous Pemphigoid–Associated S aureus Increases Protease Activity from Keratinocytes and Promotes Unique BP180 Cleavage

Candida albicans Enhances Protease Activity and Activates MyD88‐Dependent IL‐1β Production in Human Keratinocytes

CLDN1 knock out keratinocytes as a model to investigate multiple skin disorders

Differentiation of keratinocytes or exposure to type 2 cytokines diminishes S. aureus internalization

Electrical Impedance Spectroscopy Quantifies Skin Barrier Function in Organotypic In Vitro Epidermis Models

Stage of Keratinocyte Differentiation Is a Key Determinant of Viral Susceptibility in Human Skin

JAK Signaling Is Critically Important in Cytokine-Induced Viral Susceptibility of Keratinocytes

S. aureus virulence factors decrease epithelial barrier function and increase susceptibility to viral infection

Supply Chain Disruptions during COVID-19 Pandemic Uncover Differences in Keratinocyte Culture Media

Monkeypox: Considerations as a New Pandemic Looms

Conditions That Simulate the Environment of Atopic Dermatitis Enhance Susceptibility of Human Keratinocytes to Vaccinia Virus

An increase in glycoprotein concentration on extracellular virions dramatically alters vaccinia virus infectivity and pathogenesis without impacting immunogenicity

Characterization of Human Keratinocyte Cell Lines for Barrier Studies

Research Techniques Made Simple: Delivery of the CRISPR/Cas9 Components into Epidermal Cells

Peptides Derived from the Tight Junction Protein CLDN1 Disrupt the Skin Barrier and Promote Responsiveness to an Epicutaneous Vaccine

Collaborators (5)

Hector Aguilar-Carreno

Benjamin Miller

Lisa Beck

Jos Smits

H. Mark Kenney

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