Matthew Stachler

Assistant Professor

University of California, San Francisco

United States

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Matthew Stachler is an Assistant Professor at the University of California San Francisco, USA. His research areas include the study of Barrett’s esophagus, cancer treatment impacts on bacterial operons, and the immune microenvironment in relation to gastrointestinal cancers. Recent articles authored by Professor Stachler emphasize the clinical implications of cellular changes in Barrett’s esophagus and the potential therapeutic targets in gastric cancer.

Articles (10)

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2025

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2024

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2023

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2023

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2023

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2023

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2023

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2022

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2022

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2021

Collaborators (3)

Ryan Hekman

Boston University

UNITED STATES

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Peter Turnbaugh

Assistant Professor

University of California, San Francisco

UNITED STATES

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Douglas Pleskow

Professor of Medicine

Harvard University

UNITED STATES

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Matthew Stachler

Research Interests

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Articles (10)

Expansion of a bacterial operon during cancer treatment ameliorates fluoropyrimidine toxicity

Refining definitions of Barrett’s oesophagus to improve clinical resource utilisation

Degree of crypt atypia correlates with progression to high‐grade dysplasia/adenocarcinoma in non‐dysplastic Barrett's oesophagus

Interleukin-33 as a Potential Therapeutic Target in Gastric Cancer Patients: Current Insights

Bridging the Gap between Diseases of the Stomach and Lower Esophagus

Feasibility Study Utilizing NanoString’s Digital Spatial Profiling (DSP) Technology for Characterizing the Immune Microenvironment in Barrett’s Esophagus Formalin-Fixed Paraffin-Embedded Tissues

Does radiofrequency ablation of the lower oesophagus allow for clonal expansion of highly mutated neosquamous epithelium?

Pilot Study Showing Feasibility of Phosphoproteomic Profiling of Pathway-Level Molecular Alterations in Barrett’s Esophagus

Today’s Mistakes and Tomorrow’s Wisdom in Development and Use of Biomarkers for Barrett’s Esophagus

Abnormal TP53 predicts risk of progression in patients with Barrett’s esophagus regardless of a diagnosis of dysplasia.

Collaborators (3)

Ryan Hekman

Peter Turnbaugh

Douglas Pleskow

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