Max Löhning

University Professor

Charité - Universitätsmedizin Berlin

Germany

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About

Max Löhning is a University Professor at Charité Universitätsmedizin Berlin, Germany. His research focuses on immunology and its implications in intestinal inflammation, tumorigenesis, and osteoarthritis, as evidenced by his recent publications. Additionally, he investigates the transcriptional profiles of immune cells and their roles in viral infections and pain management.

Articles (14)

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2025

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2022

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2022

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2022

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2022

Collaborators (15)

Sanjiv Luther

Full Professor

University of Lausanne

SWITZERLAND

Max Löhning

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Articles (14)

The IL-22-oncostatin M axis promotes intestinal inflammation and tumorigenesis.

The impact of disease and species differences on the intestinal CLCA4 gene expression.

A type 1 immunity-restricted promoter of the IL−33 receptor gene directs antiviral T-cell responses

Plasticity and lineage commitment of individual TH 1 cells are determined by stable T-bet expression quantities.

A buprenorphine depot formulation provides effective sustained post-surgical analgesia for 72 h in mouse femoral fracture models.

NOS inhibition reverses TLR2-induced chondrocyte dysfunction and attenuates age-related osteoarthritis.

The alarmin interleukin-33 promotes the expansion and preserves the stemness of Tcf-1+ CD8+ T cells in chronic viral infection.

Insights into osteoarthritis development from single-cell RNA sequencing of subchondral bone

Optimization of chondrocyte isolation from human articular cartilage to preserve the chondrocyte transcriptome

Dissecting the dynamic transcriptional landscape of early T helper cell differentiation into Th1, Th2, and Th1/2 hybrid cells.

Mechanical forces couple bone matrix mineralization with inhibition of angiogenesis to limit adolescent bone growth.

CD39 and CD326 Are Bona Fide Markers of Murine and Human Plasma Cells and Identify a Bone Marrow Specific Plasma Cell Subpopulation in Lupus.

Menstrual blood-derived mesenchymal stromal cells efficiently ameliorate experimental autoimmune encephalomyelitis by inhibiting T cell activation in mice.

[The DRFZ-a pioneer in research on the interaction between immune and stromal cells during de- and regeneration of the musculoskeletal system].

Collaborators (15)

Sanjiv Luther

Tazio Maleitzke

Robert Klopfleisch

Georgia Katsoula

Frank Konietschke

Thorsten Schinke

Daniel Pinschewer

Peter Reuther

Tobias Winkler

Florian Kreppel

Ahmed N. Hegazy

Tuan Anh Le

Jay Roodselaar

Frederik Heinrich

Frank Buttgereit

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