Mayukh Banerjee

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Assistant Professor

University of Louisville

United States

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Dr. Mayukh Banerjee is an Assistant Professor at the University of Louisville, USA. His research predominantly focuses on the molecular mechanisms of carcinogenesis related to chronic arsenic exposure, as well as the effects of environmental toxins on cellular processes. Recent publications highlight his investigations into alternative splicing in skin carcinogenesis, DNA damage response in breast cancer, and the impact of microplastics on metabolic pathways.

Recent Grants

Grant: Close

APC11 is a novel target for arsenic-mediated zinc displacement leading to cell cycle disruption

Open Date: 2020-10-01

Close Date: 2021-09-01

Grant: Close

Microbial metabolites protect against arsenic induced gut barrier dysfunction

Open Date: 2020-09-01

Close Date: 2021-08-01

Grant: Close

Alternative splicing in arsenical skin carcinogenesis

Open Date: 2020-07-14

Close Date: 2022-06-30

Grant: Close

Mechanism for arsenic induced carcinogenesis

Open Date: 2017-08-01

Close Date: 2022-07-31

Grant: Close

The role of MRP4 in presenting arsenic-induced toxicity and carcinogenesis

Open Date: 2014-09-01

Close Date: 2016-12-31

Articles (23)

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2021

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2021

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2021

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2020

Collaborators (5)

Theodore Kalbfleisch

Associate Professor

University of Kentucky

UNITED STATES

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J. Christopher States

Professor

University of Louisville

UNITED STATES

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Jingjing Zhao

University of Louisville

UNITED STATES

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Elaine Leslie

University of Alberta

CANADA

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Michael Merchant

Professor

University of Louisville

UNITED STATES

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Mayukh Banerjee

Research Interests

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About

Recent Grants

Articles (23)

Retraction notice to “Arsenic-induced mitochondrial instability leading to programmed cell death in the exposed individuals" [Toxicology 246 (2008) 101–111]

Chronic arsenic exposure and hsa-miR-186 overexpression causes transcriptome-wide differential alternative splicing contributing to skin carcinogenesis in human HaCaT cell line

DNA damage response inhibition is an early event in cadmium-induced breast carcinogenesis

Perfluorooctane sulfonate exposure and alcohol-associated liver disease severity in a mouse chronic-binge ethanol feeding model

Dysregulation of mRNA expression by hsa-miR-186 overexpression in arsenic-induced skin carcinogenesis

Obesogenic polystyrene microplastic exposures disrupt the gut-liver-adipose axis

Chronic arsenic exposure suppresses proteasomal and autophagic protein degradation

Chronic arsenic exposure induces malignant transformation of human HaCaT cells through both deterministic and stochastic changes in transcriptome expression

Differential Selectivity of Human and Mouse ABCC4/Abcc4 for Arsenic Metabolites

Chronic arsenic exposure induces malignant transformation of human HaCaT cells through both deterministic and stochastic changes in transcriptome expression

Altered splicing factor and alternative splicing events in a mouse model of diet- and polychlorinated biphenyl-induced liver disease

Association of ABCC4 G559T single nucleotide polymorphism with arsenic-induced precancerous hyperkeratosis

Clonal variability in chromosomal instability as a potential driver in the acquisition of tumorigenic phenotype in chronic arsenic-exposed and hsa-miR-186 overexpressing human keratinocytes

miR-186 induces tetraploidy in arsenic exposed human keratinocytes

Zinc supplementation prevents mitotic accumulation in human keratinocyte cell lines upon environmentally relevant arsenic exposure.

Zinc supplementation prevents mitotic accumulation in human keratinocyte cell lines upon environmentally relevant arsenic exposure

Temporal Modulation of Differential Alternative Splicing in HaCaT Human Keratinocyte Cell Line Chronically Exposed to Arsenic for up to 28 Wk

Zinc supplementation prevents arsenic-induced dysregulation of ZRANB2 splice function

Chronic arsenic exposure suppresses ATM pathway activation in human keratinocytes

Delineating the Effects of Passaging and Exposure in a Longitudinal Study of Arsenic-Induced Squamous Cell Carcinoma in a HaCaT Cell Line Model

miRNA dysregulation is an emerging modulator of genomic instability

Dynamic alteration in miRNA and mRNA expression profiles at different stages of chronic arsenic exposure-induced carcinogenesis in a human cell culture model of skin cancer

Arsenite Exposure Displaces Zinc from ZRANB2 Leading to Altered Splicing.

Collaborators (5)

Theodore Kalbfleisch

J. Christopher States

Jingjing Zhao

Elaine Leslie

Michael Merchant

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