Michael Bader

Professor

Charité - Universitätsmedizin Berlin

Germany

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Professor Michael Bader is a faculty member at Charite Universitätsmedizin Berlin, Germany. His research primarily focuses on the biochemical mechanisms of serotonin synthesis and its implications for various health conditions, including pulmonary arterial hypertension and renal damage. Recent publications highlight his work on innovative inhibitor development and the role of angiotensin in neurodevelopment and growth. His contributions to the field are essential for understanding and treating conditions related to serotonin and angiotensin signaling pathways.

Articles (20)

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2025

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2021

Collaborators (15)

Camila Guimarães Moreira Zimmer

Head of Preclinical Research and Development

UNITED STATES

Michael Bader

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Articles (20)

Selectivity and Safety Characterization of a Xanthine–Imidazothiazole Lead Structure: a Novel Tryptophan Hydroxylase Inhibitor of Peripheral Serotonin Synthesis

Inhibiting Serotonin Synthesis for the Treatment of Pulmonary Arterial Hypertension

Renal damage-induced hepcidin accumulation contributes to anemia in angiotensinogen-deficient mice

Angiotensin-(1-12): Does It Exist? A Critical Evaluation in Humans, Rats, and Mice

Role of kinin receptors in skin pigmentation

TPT‐004, a Next‐Generation Inhibitor of Tryptophan Hydroxylase, Ameliorates Pulmonary Arterial Hypertension in Rats

Serotonin Transporter-dependent Histone Serotonylation in Placenta Contributes to the Neurodevelopmental Transcriptome

Socio-affective communication in Tph2-deficient rat pups: communal nesting aggravates growth retardation despite ameliorating maternal affiliation deficits

Renin Is Essential for Angiotensin II Formation in the Brain

Normal male fertility in a mouse model of KPNA2 deficiency

Angiotensin-(1−7) mediated calcium signalling by MAS

Evidence in favor of the essentiality of human cell membrane-bound ACE2 and against soluble ACE2 for SARS-CoV-2 infectivity

Myogenic Vasoconstriction Requires Canonical G<sub>q/11</sub> Signaling of the Angiotensin II Type 1 Receptor

Oxidative RNA Damage in the Pathogenesis and Treatment of Type 2 Diabetes

Peripheral Serotonin Deficiency Affects Anxiety-like Behavior and the Molecular Response to an Acute Challenge in Rats

Role of Gpnmb in atherosclerosis of female mice

Serotonin limits generation of chromaffin cells during adrenal organ development

Tph2 Gene Expression Defines Ethanol Drinking Behavior in Mice

(Pro)renin Receptor Inhibition Reduces Plasma Cholesterol and Triglycerides but Does Not Attenuate Atherosclerosis in Atherosclerotic Mice.

Diabetic pregnancy as a novel risk factor for cardiac dysfunction in the offspring-the heart as a target for fetal programming in rats.

Collaborators (15)

Camila Guimarães Moreira Zimmer

Michaela Golic

Jinye Dai

Aliia Murtazina

Vanessa Monteil

Dirk Pleimes

Dominik N Muller

Valentina Mosienko

Kristin Kräker

Edgar Specker

Markéta Tesařová

Florian Herse

Dien Ye

Judith Homberg

Francesca Calabrese

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