MICHAEL GOLDING

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Associate Professor

Texas A&M University

United States

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Michael Golding is an Associate Professor at Texas A&M University in the United States. His research focuses on the impacts of paternal alcohol exposure on offspring, particularly in areas such as mitochondrial dysfunction, craniofacial development, and epigenetic programming. His recent publications include studies on the lasting effects of parental alcohol consumption and its implications for offspring health and development.

Recent Grants

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HERITABLE, EPIGENETIC EFFECTS OF PATERNAL ALCOHOL USE ON FASD PHENOTYPES

Open Date: 2020-06-15

Close Date: 2025-03-31

Grant: Close

To explore the mechanisms of paternal contributions to fetal alcohol spectrum disorders.

Open Date: 2019-06-01

Close Date: 2022-05-31

Grant: Close

Altered genomic imprinting as a basis for FASD placental growth defects

Open Date: 2014-09-10

Close Date: 2016-08-31

Grant: Close

Measuring the impact of prenatal alcohol exposure on the fetal epigenome

Open Date: 2010-09-30

Close Date: 2013-08-31

Grant: Close

Measuring the impact of prenatal alcohol exposure on the fetal epigenome

Open Date: 2010-09-30

Close Date: 2013-08-31

Articles (15)

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2024

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2024

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2024

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2023

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2023

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2022

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2022

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2022

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2022

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2022

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2021

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2021

Collaborators (4)

Renyi Zhang

University Distinguished Professor and Holder of Harold J. Haynes Endowed Chair in Geosciences

Texas A&M University

UNITED STATES

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Claude Robert

Professeur titulaire

Laval University

CANADA

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Natalie M. Johnson

Texas A&M University

UNITED STATES

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Aline Rodrigues Hoffmann

Associate Professor

University of Florida

UNITED STATES

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MICHAEL GOLDING

Research Interests

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About

Recent Grants

Articles (15)

Alterations in sperm RNAs persist after alcohol cessation and correlate with epididymal mitochondrial dysfunction

Parental Alcohol Exposures Associate with Lasting Mitochondrial Dysfunction and Accelerated Aging in a Mouse Model.

Chronic paternal alcohol exposures induce dose-dependent changes in offspring craniofacial shape and symmetry.

Maternal 129S1/SvImJ background attenuates the placental phenotypes induced by chronic paternal alcohol exposure.

Teratogenesis and the epigenetic programming of congenital defects: Why paternal exposures matter

Preconception paternal ethanol exposures induce alcohol-related craniofacial growth deficiencies in fetal offspring

Preconception paternal alcohol exposure decreases IVF embryo survival and pregnancy success rates in a mouse model

Paternal alcohol exposure and dental-facial anomalies in offspring. Reply.

NRF2-Dependent Placental Effects Vary by Sex and Dose following Gestational Exposure to Ultrafine Particles.

Paternal epigenetic influences on placental health and their impacts on offspring development and disease.

Paternal alcohol exposures program intergenerational hormetic effects on offspring fetoplacental growth.

Alcohol induced increases in sperm Histone H3 lysine 4 trimethylation correlate with increased placental CTCF occupancy and altered developmental programming.

Chromatin alterations during the epididymal maturation of mouse sperm refine the paternally inherited epigenome.

Maternal background alters the penetrance of growth phenotypes and sex‐specific placental adaptation of offspring sired by alcohol‐exposed males

Gestational Exposure to Ultrafine Particles Reveals Sex- and Dose-Specific Changes in Offspring Birth Outcomes, Placental Morphology, and Gene Networks.

Collaborators (4)

Renyi Zhang

Claude Robert

Natalie M. Johnson

Aline Rodrigues Hoffmann

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