Michael Grusch

Assoc. Prof.

Medical University of Vienna

Austria

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About

Michael Grusch is an Associate Professor at the Medical University of Vienna, Austria. His research primarily focuses on cancer biology, with recent articles addressing topics such as mesothelioma, cell motility regulation, and the effects of fatty acid metabolism on cancer cell behavior. He also investigates the genetic and environmental factors influencing DNA damage in occupational exposure scenarios.

Articles (15)

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2025

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2025

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2024

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2024

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2024

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2023

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2023

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2020

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2020

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2020

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2020

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2020

Collaborators (14)

Andrew Burgess

The University of Queensland Diamantina Institute

AUSTRALIA

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Seema Gupta

Research Assistant Professor

Georgetown University

UNITED STATES

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Joachim Widder

Medical University of Vienna

AUSTRIA

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Marina Kennerson

Principal Hospital Scientist / Professor

AUSTRALIA

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Glen Reid

University of Otago

NEW ZEALAND

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Christopher Gerner

Full Professor

AUSTRIA

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Christian Kowol

Associate Professor

Universität Wien

AUSTRIA

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Stefan Watzka

Provisional Chairman/Associate Professor of Surgery

AUSTRIA

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Walter Berger

AUSTRIA

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Astrid Slany

Universität Wien

AUSTRIA

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Enikö Kallay

Assoc. Prof.

Medical University of Vienna

AUSTRIA

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Balazs Dome

Medical University of Vienna

AUSTRIA

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Balazs Hegedus

University Hospital Essen

GERMANY

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Karin Schelch

Medical University of Vienna

AUSTRIA

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Michael Grusch

Research Interests

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Articles (15)

The Mesothelioma Systemic Inflammation Score Is Independently Associated with Overall Survival and Predicts Benefit of Multimodality Treatment in Pleural Mesothelioma.

The micronucleus assay in exfoliated buccal cells for occupational exposure studies: an overview

ERK1/2 mitogen‐activated protein kinase dimerization is essential for the regulation of cell motility

High circulating activin A plasma levels are associated with tumour stage and poor survival in treatment-naive lung squamous cell cancer patients.

Targeting endogenous fatty acid synthesis stimulates the migration of ovarian cancer cells to adipocytes and promotes the transport of fatty acids from adipocytes to cancer cells

YB-1 regulates mesothelioma cell migration via snail but not EGFR, MMP1, EPHA5 or PARK2

Impact of mobile phone-specific electromagnetic fields on DNA damage caused by occupationally relevant exposures: results of ex vivo experiments with peripheral blood mononuclear cells from different demographic groups

Primary and hTERT-Transduced Mesothelioma-Associated Fibroblasts but Not Primary or hTERT-Transduced Mesothelial Cells Stimulate Growth of Human Mesothelioma Cells

Influence of the Fatty Acid Metabolism on the Mode of Action of a Cisplatin(IV) Complex with Phenylbutyrate as Axial Ligands

Circulating FGF18 is decreased in pleural mesothelioma but not correlated with disease prognosis

Shifting the Immune-Suppressive to Predominant Immune-Stimulatory Radiation Effects by SBRT-PArtial Tumor Irradiation Targeting HYpoxic Segment (SBRT-PATHY)

Activin A: an emerging target for improving cancer treatment?

YB-1 Knockdown Inhibits the Proliferation of Mesothelioma Cells through Multiple Mechanisms

Telomerase Reverse Transcriptase Promoter Mutations Identify a Genomically Defined and Highly Aggressive Human Pleural Mesothelioma Subgroup

P38 MAPK Promotes Migration and Metastatic Activity of BRAF Mutant Melanoma Cells by Inducing Degradation of PMCA4b

Collaborators (14)

Andrew Burgess

Seema Gupta

Joachim Widder

Marina Kennerson

Glen Reid

Christopher Gerner

Christian Kowol

Stefan Watzka

Walter Berger

Astrid Slany

Enikö Kallay

Balazs Dome

Balazs Hegedus

Karin Schelch

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