Michael Murrell

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Associate Professor

Yale University

United States

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About

Michael Murrell is an Associate Professor at Yale University. His research primarily focuses on the dynamics of the cell cytoskeleton, active matter, and the mechanical properties of actin networks. Recent publications include studies on energy localization in cellular structures, actomyosin dynamics, and the role of cofilin in enhancing filament flexibility. His work aims to deepen understanding of mechanical power during cellular processes and the physical principles governing cell behavior.

Recent Grants

Grant: Close

Energetic Costs to Regulation in the Excitable Cytoskeleton

Open Date: 2022-01-01

Close Date: 2024-01-01

Grant: Close

Collaborative Research: International Physics of Living Systems Graduate Research Network

Open Date: 2021-09-15

Close Date: 2026-08-31

Grant: Close

Molecular control of cortical homeostasis and cell polarization

Open Date: 2018-05-01

Close Date: 2021-04-30

Grant: Close

Universal Roles of Force Generation and Transmission in Biological Systems

Open Date: 2017-01-01

Close Date: 2023-01-01

Grant: Close

Mechanisms of Force Sensing in Adherent Cells as Inspiration for New Materials

Open Date: 2016-01-01

Close Date: 2022-01-01

Articles (16)

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2025

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2023

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2023

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2023

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2023

Collaborators (7)

Daniel Seara

University of Chicago

UNITED STATES

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Margaret L. Gardel

University of Chicago

UNITED STATES

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David Calderwood

Yale University School of Medicine

UNITED STATES

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Xingbo Shang

University of California, Los Angeles

UNITED STATES

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Jesse Rinehart

Associate Professor

Yale University

UNITED STATES

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Eric Robert Dufresne

Professor

Cornell University

UNITED STATES

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martin schwartz

Professor

Yale University School of Medicine

UNITED STATES

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Michael Murrell

Research Interests

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About

Recent Grants

Articles (16)

Feedback between F-actin organization and active stress governs criticality and energy localization in the cell cytoskeleton

Illuminating active matter by harnessing light for modular flow control

Elastocapillary effects determine early matrix deformation by glioblastoma cell spheroids

Energy partitioning in the cell cortex

Composite branched and linear F-actin maximize myosin-induced membrane shape changes in a biomimetic cell model

Active tension and membrane friction mediate cortical flows and blebbing in a model actomyosin cortex

Confinement induces internal flows in adherent cell aggregates

F-actin architecture determines the conversion of chemical energy into mechanical work

Author Correction: Mechanical power is maximized during contractile ring-like formation in a biomimetic dividing cell model

Cofilin-Mediated Filament Softening and Crosslinking Counterbalance to Enhance Actin Network Flexibility

Mechanical power is maximized during contractile ring-like formation in a biomimetic dividing cell model

Cofilin-mediated actin filament network flexibility facilitates 2D to 3D actomyosin shape change

Author Correction: Membrane tension induces F-actin reorganization and flow in a biomimetic model cortex

Intracellular tension sensor reveals mechanical anisotropy of the actin cytoskeleton

Actin and Microtubules Position Stress Granules

SPAK-dependent co-transporter activity mediates capillary adhesion and pressure during glioblastoma migration in confined spaces

Collaborators (7)

Daniel Seara

Margaret L. Gardel

David Calderwood

Xingbo Shang

Jesse Rinehart

Eric Robert Dufresne

martin schwartz

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