Michel BOURGUIGNON

Professor Emeritus

Université Paris-Saclay

France

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Michel Bourguignon is a Professor Emeritus at the University of Paris-Saclay, France. His research focuses on the cellular and molecular responses to ionizing radiation, emphasizing the role of the ATM protein in various conditions, including cancers and genetic disorders. Recent publications explore topics such as accelerated aging effects in space, the influence of gamma radiation on DNA mutability, and potential early diagnostic markers for Alzheimer's disease.

Articles (10)

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2025

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2024

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2024

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2024

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2024

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2024

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2023

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2023

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2023

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2023

Collaborators (3)

EYMERIC LE REUN

Assistant Professor of Radiation Oncology

Université Paris Cité

FRANCE

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Dorota Slonina

Ph.D, DSc, Associate Professor

Maria Sklodowska-Curie National Research Institute of Oncology

POLAND

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Thibaud Mathis

FRANCE

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Michel BOURGUIGNON

Research Interests

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Articles (10)

Skin Fibroblasts from Individuals Self-Diagnosed as Electrosensitive Reveal Two Distinct Subsets with Delayed Nucleoshuttling of the ATM Protein in Common

Influence of the Nucleo-Shuttling of the ATM Protein on the Response of Skin Fibroblasts from Marfan Syndrome to Ionizing Radiation

Accelerated Aging Effects Observed In Vitro after an Exposure to Gamma-Rays Delivered at Very Low and Continuous Dose-Rate Equivalent to 1–5 Weeks in International Space Station

Prediction of Cancer Proneness under Influence of X-rays with Four DNA Mutability and/or Three Cellular Proliferation Assays

When Chromatin Decondensation Affects Nuclear γH2AX Foci Pattern and Kinetics and Biases the Assessment of DNA Double-Strand Breaks by Immunofluorescence

When DNA Mutations Interplay with Cellular Proliferation: A Narrative History of Theories of Carcinogenesis

The Radiobiological Characterization of Human and Porcine Lens Cells Suggests the Importance of the ATM Kinase in Radiation-Induced Cataractogenesis

Toward an Early Diagnosis for Alzheimer’s Disease Based on the Perinuclear Localization of the ATM Protein

X-rays-Induced Bystander Effect Consists in the Formation of DNA Breaks in a Calcium-Dependent Manner: Influence of the Experimental Procedure and the Individual Factor

Response of Fibroblasts from Menkes’ and Wilson’s Copper Metabolism-Related Disorders to Ionizing Radiation: Influence of the Nucleo-Shuttling of the ATM Protein Kinase

Collaborators (3)

EYMERIC LE REUN

Dorota Slonina

Thibaud Mathis

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