Mitchell Fane

Assistant Professor

Fox Chase Cancer Center

United States

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About

Dr. Mitchell Fane is an Assistant Professor at Fox Chase Cancer Center in the United States. His research primarily focuses on melanoma therapy resistance, cancer progression related to aging, and the role of cellular signaling pathways. Recent publications highlight his investigations into cellular mechanisms that drive melanoma cell invasion and the impact of the tumor microenvironment on cancer dynamics.

Articles (15)

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2024

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2020

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2020

Collaborators (9)

Nikolas Haass

Professorial Research Fellow/Professor for Cutaneous Oncology

University of Queensland

AUSTRALIA

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Sarah Bayatpour

Virginia Commonwealth University

UNITED STATES

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Yogesh Goyal

Assistant Professor

Northwestern University

UNITED STATES

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Marie Webster

Assistant Professor

Lankenau Institute for Medical Research

UNITED STATES

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Daniel Zabransky

Johns Hopkins University

UNITED STATES

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Jacinta Simmons

Queensland University of Technology

AUSTRALIA

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Laura Kim

University of Pennsylvania Perelman School of Medicine

UNITED STATES

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Sydney Shaffer

Assistant Professor

University of Pennsylvania

UNITED STATES

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Todd Armstrong

Johns Hopkins University

UNITED STATES

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Mitchell Fane

Research Interests

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About

Articles (15)

Fibroblasts in the Aged Pancreas Drive Pancreatic Cancer Progression

Age-Related Increases in IGFBP2 Increase Melanoma Cell Invasion and Lipid Synthesis

Determinants of resistance and response to melanoma therapy

Author Correction: Disrupting cellular memory to overcome drug resistance

Disrupting cellular memory to overcome drug resistance

Stromal changes in the aged lung induce an emergence from melanoma dormancy

ClampFISH 2.0 enables rapid, scalable amplified RNA detection in situ

Reciprocal regulation of BRN2 and NOTCH1/2 signaling synergistically drives melanoma cell migration and invasion

Genetic screening for single-cell variability modulators driving therapy resistance.

Myeloid-Derived Suppressor Cells Are a Major Source of Wnt5A in the Melanoma Microenvironment and Depend on Wnt5A for Full Suppressive Activity.

sFRP2 Supersedes VEGF as an Age-related Driver of Angiogenesis in Melanoma, Affecting Response to Anti-VEGF Therapy in Older Patients.

Stromal Changes In The Aged Lung Induce An Emergence From Melanoma Dormancy

Changes in Aged Fibroblast Lipid Metabolism Induce Age-Dependent Melanoma Cell Resistance to Targeted Therapy via the Fatty Acid Transporter FATP2

Paradoxical Role for Wild-Type p53 in Driving Therapy Resistance in Melanoma

Erratum: Paradoxical Role for Wild-Type p53 in Driving Therapy Resistance in Melanoma (Molecular Cell (2020) 77(3) (633–644.e5), (S109727651930838X), (10.1016/j.molcel.2019.11.009))

Collaborators (9)

Nikolas Haass

Sarah Bayatpour

Yogesh Goyal

Marie Webster

Daniel Zabransky

Jacinta Simmons

Laura Kim

Sydney Shaffer

Todd Armstrong

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