Moe Mahjoub

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Assistant Professor

Washington University in St. Louis

United States

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About

Moe Mahjoub is an Assistant Professor at Washington University in St. Louis, USA. His research focuses on polycystic kidney disease, highlighting aspects such as multiomics profiling, cystogenesis, and the role of specific genes in kidney development and function. Recent publications include studies on the implications of DLG1 and Cep120 in kidney health, as well as advanced imaging techniques for analyzing kidney structures.

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CENTROSOME DYSFUNCTION IN POLYCYSTIC KIDNEY DISEASE

Open Date: 2015-12-01

Close Date: 2020-11-30

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Collaborators (5)

James Thompson

University of Southampton

UNITED KINGDOM

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sanjay jain

Washington University in St. Louis

UNITED STATES

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Hui Zong

UNITED STATES

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Vito Mennella

Associate Professor/Reader

Queen Mary University of London

UNITED KINGDOM

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Eirini Kefalogianni

Assistant Professor of Medicine

Washington University in Saint Louis School of Medicine

UNITED STATES

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Moe Mahjoub

Research Interests

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About

Recent Grants

Articles (19)

Inhibiting centrosome clustering reduces cystogenesis and improves kidney function in autosomal dominant polycystic kidney disease

Cep120 is essential for kidney stromal progenitor cell growth and differentiation

DLG1 functions upstream of SDCCAG3 and IFT20 to control ciliary targeting of polycystin-2

Multiomics profiling of mouse polycystic kidney disease progression at a single-cell resolution

DLG1 functions upstream of SDCCAG3 and IFT20 to control ciliary targeting of polycystin-2

Multi-omics profiling of mouse polycystic kidney disease progression at a single cell resolution

The Heterotaxy Gene <i>CCDC11</i> Is Important for Cytokinesis via RhoA Regulation

Ultrastructure expansion microscopy (U-ExM) of mouse and human kidneys for analysis of subcellular structures

Ultrastructure expansion microscopy (U-ExM) of mouse and human kidneys for analysis of subcellular structures

Cep120 is essential for kidney stromal progenitor cell growth and differentiation

Aberrant centrosome biogenesis disrupts nephron and collecting duct progenitor growth and fate resulting in fibrocystic kidney disease

Aberrant centrosome biogenesis disrupts nephron and collecting duct progenitor growth and fate resulting in fibrocystic kidney disease

Aberrant centrosome biogenesis disrupts nephron progenitor cell renewal and fate resulting in fibrocystic kidney disease

GEMC1 and MCIDAS interactions with SWI/SNF complexes regulate the multiciliated cell-specific transcriptional program

Impaired centrosome biogenesis in kidney stromal progenitors reduces abundance of interstitial lineages and accelerates injury-induced fibrosis

The effect of<i> Dnaaf5</i> gene dosage on primary ciliary dyskinesia phenotypes

RNA-Seq identifies genes whose proteins are upregulated during syncytia development in murine C2C12 myoblasts and human BeWo trophoblasts

Mosaic Analysis with Double Markers reveals IGF1R function in granule cell progenitors during cerebellar development

Super-Resolution Microscopy and FIB-SEM Imaging Reveal Parental Centriole-Derived, Hybrid Cilium in Mammalian Multiciliated Cells

Collaborators (5)

James Thompson

sanjay jain

Hui Zong

Vito Mennella

Eirini Kefalogianni

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