Nadia Stefanova

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Professor of Translational Neurodegeneration Research

Medical University of Innsbruck

Austria

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About

Professor Nadia Stefanova is a prominent researcher in Translational Neurodegeneration Research at the Medical University of Innsbruck, Austria. Her recent work focuses on the molecular mechanisms underlying multiple system atrophy and Parkinson's disease, exploring topics such as transcriptomic dysregulation and neuroinflammation. She has contributed significantly to the understanding of α-synuclein pathology through innovative models and genomic analyses.

Recent Grants

Grant: Close

Alpha-synuclein: a pathogenic trigger and interventional target in MSA

Open Date: 2015-01-01

Close Date: 2019-01-01

Grant: Close

Alpha-Synuclein and ligodendroglia in MSA pathogenesis

Open Date: 2013-01-01

Close Date: 2016-12-31

Grant: Close

MSA and Neuroinflammation

Open Date: 2007-08-01

Close Date: 2010-07-31

Grant: Close

Signal Processing in Neurons

Open Date:

Close Date:

Articles (18)

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2025

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2021

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2021

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2021

Collaborators (6)

S. Stephen Yi

University of Texas at Austin

UNITED STATES

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Géraldine Gelders

Janssen Pharmaceutica NV

BELGIUM

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Zichen (Zachary) Wang

University of California, San Diego

UNITED STATES

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Per Borghammer

Aarhus University

DENMARK

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MARIA TERESA PELLECCHIA

Associate Professor of Neurology

University of Salerno

ITALY

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Walter Struhal

Karl Landsteiner University of Health Sciences

AUSTRIA

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Nadia Stefanova

Research Interests

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About

Recent Grants

Articles (18)

Nigral Neuroinflammation and Dopaminergic Neurons in Parkinson's Disease and Atypical Parkinsonisms

Transcriptional dysregulation in the cerebellum triggered by oligodendroglial α-synucleinopathy: insights from a transgenic mouse into the early disease mechanisms of MSA.

Rapid iPSC inclusionopathy models shed light on formation, consequence, and molecular subtype of α-synuclein inclusions.

Gregor K. Wenning: a brilliant mind

Genome sequence analyses identify novel risk loci for multiple system atrophy

Transcriptomic insights into multiple system atrophy from a PLP-α-synuclein transgenic mouse model

Host oligodendrogliopathy and α-synuclein strains dictate disease severity in multiple system atrophy

A Mouse Model of Multiple System Atrophy: Bench to Bedside

Epsin2, a novel target for multiple system atrophy therapy via α-synuclein/FABP7 propagation

Multiple system atrophy: at the crossroads of cellular, molecular and genetic mechanisms

Spreading of Aggregated α-Synuclein in Sagittal Organotypic Mouse Brain Slices

ATH434 Reduces α‐Synuclein‐Related Neurodegeneration in a Murine Model of Multiple System Atrophy

The Compound ATH434 Prevents Alpha-Synuclein Toxicity in a Murine Model of Multiple System Atrophy.

The Concept of α-Synuclein Strains and How Different Conformations May Explain Distinct Neurodegenerative Disorders.

Current experimental disease-modifying therapeutics for multiple system atrophy

Is Multiple System Atrophy a Prion-like Disorder?

Therapeutic potential of iron modulating drugs in a mouse model of multiple system atrophy.

Toll-like receptor 4 deficiency facilitates α-synuclein propagation and neurodegeneration in a mouse model of prodromal Parkinson's disease.

Collaborators (6)

S. Stephen Yi

Géraldine Gelders

Zichen (Zachary) Wang

Per Borghammer

MARIA TERESA PELLECCHIA

Walter Struhal

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