Natalie Gassman

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Associate Professor

The University of Alabama

United States

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About

Dr. Natalie Gassman is an Associate Professor at The University of Alabama at Birmingham, USA. Her research areas encompass physiological responses to environmental stressors, molecular damage repair, and cancer biology, with recent publications exploring glucocorticoid responses in zebra finches, the effects of food restriction during offspring provisioning, and the genotoxicity of dihydroxyacetone. Dr. Gassman's work contributes to understanding DNA damage, cellular responses, and potential therapeutic strategies in oncology.

Recent Grants

Grant: Close

DNA Damage as a Predictive Biomarker for Immunotherapy Response in Ovarian Cancer

Open Date: 2021-07-15

Close Date: 2024-12-14

Grant: Close

Dihydroxyacetone exposure induces metabolic reprogramming and mitochondrial dysfunction

Open Date: 2021-01-01

Close Date: 2025-10-31

Grant: Close

Multi-Well Extracellular Flux Analyzer for Mitochondrial Stress Arrays

Open Date: 2020-06-01

Close Date: 2021-05-31

Grant: Close

Co-detection of DNA adducts and methylation sites as predictive biomarkers for exposures

Open Date: 2017-09-01

Close Date: 2019-08-31

Grant: Close

Bisphenol A modulation of DNA repair triggered by environmental genotoxic stress

Open Date: 2015-12-01

Close Date: 2018-11-30

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Collaborators (9)

Samuel Trammell

Københavns Universitet

DENMARK

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Yuval Ebenstein

ISRAEL

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Dieter Liebhart

University of Veterinary Medicine Vienna

AUSTRIA

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Jeffrey A. Jones

Baylor College of Medicine

UNITED STATES

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Jacob Raber

Professor

UNITED STATES

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marie migaud

Professor and Director

University of South Alabama

UNITED STATES

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Milton McAllister

Associate Professor

The University of Adelaide - Roseworthy Campus

AUSTRALIA

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Glen Kisby

Emeritus Professor of Pharmacology

Western University of Health Sciences

UNITED STATES

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Farhid Hemmatzadeh

Associate Professor

University of Adelaide

AUSTRALIA

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Natalie Gassman

Research Interests

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About

Recent Grants

Articles (20)

Developmental Heat Conditioning Alters the Adult Glucocorticoid Response and DNA Damage While Mediating Lifespan Variation in Zebra Finches (Taeniopygia Castanotis)

Maintenance of Physiological and Molecular Processes Through Food Restriction During Offspring Provisioning in the Adult Zebra Finch

Physiological Responses Vary with Molecular Damage Outcomes after Recovery from Constant Light

PARP inhibitor response is enhanced in prostate cancer when XRCC1 expression is reduced

Novel Techniques for Mapping DNA Damage and Repair in the Brain

A tipping point in dihydroxyacetone exposure: mitochondrial stress and metabolic reprogramming alter survival in rat cardiomyocytes H9c2 cells

Synthesis, Detection, and Metabolism of Pyridone Ribosides, Products of NAD Overoxidation

Newcastle Disease Virus Induces Profound Lymphoid Depletion with Different Patterns of Necroptosis, Necrosis, and Oxidative DNA Damage in Bursa, Spleen, and Other Lymphoid Tissues

Acute exposure to dihydroxyacetone promotes genotoxicity and chromosomal instability in lung, cardiac, and liver cell models

Heat Shock Factor 1 Inhibition: A Novel Anti-Cancer Strategy with Promise for Precision Oncology

Dihydroxyacetone suppresses mTOR nutrient signaling and induces mitochondrial stress in liver cells

Solution Chemistry of Dihydroxyacetone and Synthesis of Monomeric Dihydroxyacetone

Glucose Increases STAT3 Activation, Promoting Sustained XRCC1 Expression and Increasing DNA Repair

Repair-Assisted Damage Detection Reveals Biological Disparities in Prostate Cancer between African Americans and European Americans

From single-molecule to genome-wide mapping of DNA lesions: repair-assisted damage detection sequencing

Cytoprotective Effect of Vitamin D on Doxorubicin-Induced Cardiac Toxicity in Triple Negative Breast Cancer

Activated STAT3 Is a Novel Regulator of the XRCC1 Promoter and Selectively Increases XRCC1 Protein Levels in Triple Negative Breast Cancer

Associations between DNA Damage and PD-L1 Expression in Ovarian Cancer, a Potential Biomarker for Clinical Response

Chemical and Biochemical Reactivity of the Reduced Forms of Nicotinamide Riboside

The Biochemical Pathways of Nicotinamide-Derived Pyridones

Collaborators (9)

Samuel Trammell

Yuval Ebenstein

Dieter Liebhart

Jeffrey A. Jones

Jacob Raber

marie migaud

Milton McAllister

Glen Kisby

Farhid Hemmatzadeh

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