Navonil De Sarkar

Assistant Professor

Medical College of Wisconsin

United States

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Navonil De Sarkar is an Assistant Professor at the Medical College of Wisconsin, USA. His research focuses on metastatic castration-resistant prostate cancer, including therapeutic implications, genomic attributes, and the efficacy of systemic therapies. His recent publications explore topics such as DNA repair deficiencies and the regulation of cancer-related genes, contributing to advancements in cancer treatment and understanding.

Articles (10)

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2022

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2021

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2021

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2021

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2021

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2021

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2021

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2021

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2021

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2020

Collaborators (3)

Gavin Ha

Assistant Professor

Fred Hutchinson Cancer Research Center

UNITED STATES

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Peter S. Nelson

University of Washington

UNITED STATES

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Keegan Korthauer

Assistant Professor

The University of British Columbia

CANADA

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Navonil De Sarkar

Research Interests

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Articles (10)

Therapeutic Implications for Intrinsic Phenotype Classification of Metastatic Castration-Resistant Prostate Cancer

Genomic attributes of homology-directed DNA repair deficiency in metastatic prostate cancer

Efficacy of systemic therapies in men with metastatic castration resistant prostate cancer harboring germline <i>ATM</i> versus <i>BRCA2</i> mutations

Reprogramming of the FOXA1 cistrome in treatment-emergent neuroendocrine prostate cancer

Concordance of DNA Repair Gene Mutations in Paired Primary Prostate Cancer Samples and Metastatic Tissue or Cell-Free DNA

Abstract LB022: Griffin: A method for nucleosome profiling and breast cancer subtype prediction from ultra-low pass whole genome sequencing of cell-free DNA

Concordance of DNA damage repair (DDR) gene mutations in paired primary and metastatic prostate cancer (PC) samples.

Selective androgen receptor modulators activate the canonical prostate cancer androgen receptor program and repress cancer growth

Regulation of CEACAM5 and Therapeutic Efficacy of an Anti-CEACAM5–SN38 Antibody–drug Conjugate in Neuroendocrine Prostate Cancer

A novel hotspot and rare somatic mutation p.A138V, at TP53 is associated with poor survival of pancreatic ductal and periampullary adenocarcinoma patients

Collaborators (3)

Gavin Ha

Peter S. Nelson

Keegan Korthauer

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