Nick van Gastel

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Associate Professor

Université catholique de Louvain

Belgium

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About

Nick van Gastel is an Associate Professor at Université Catholique de Louvain, Belgium. His research focuses on hematopoietic stem cell isolation, leukemia metabolism, and the metabolic regulation of various diseases, based on insights from stable isotope tracing and novel murine models. His recent work highlights the connections between metabolic perturbations and cancer cell apoptosis, as well as mitochondrial biogenesis in cellular contexts.

Recent Grants

Grant: Close

Targeting the Metabolic Drivers of Chemoresistance in Acute Myeloid Leukemia

Open Date: 2018-07-01

Close Date: 2020-06-01

Grant: Close

Angiogenic and osteogenic growth factors in bone tissue engineering

Open Date: 2009-01-01

Close Date: 2012-12-01

Articles (11)

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2024

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2022

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2021

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2021

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2021

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2021

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2021

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2021

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2021

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2020

Year:

2020

Collaborators (1)

Michael Wells

Assistant Professor

University of California, Los Angeles

UNITED STATES

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Nick van Gastel

Research Interests

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About

Recent Grants

Articles (11)

Optimization of pre-enrichment strategies for mouse hematopoietic stem cell isolation and metabolomic analysis

A new murine model of Barth Syndrome neutropenia links TAFAZZIN deficiency to increased ER stress induced apoptosis.

Analysis of Leukemia Cell Metabolism through Stable Isotope Tracing in Mice.

Analysis of leukemia cell metabolism through stable isotope tracing in mice

Metabolic perturbations sensitize triple-negative breast cancers to apoptosis induced by BH3 mimetics

Young haematopoietic stem cells are picky eaters

Malic enzyme 2 connects the Krebs cycle intermediate fumarate to mitochondrial biogenesis.

Imaging dynamic mTORC1 pathway activity in vivo reveals marked shifts that support time-specific inhibitor therapy in AML

Metabolic regulation of skeletal cell fate and function in physiology and disease

C9orf72 suppresses systemic and neural inflammation induced by gut bacteria

Aldehyde dehydrogenase 3a2 protects AML cells from oxidative death and the synthetic lethality of ferroptosis inducers

Collaborators (1)

Michael Wells

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