Nicolas O. Eskesen

Københavns Universitet

Denmark

Research Interests

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

Articles (8)

Year:

2024

Year:

2024

Year:

2023

Year:

2023

Year:

2022

Year:

2021

Year:

2021

Year:

2020

Collaborators (15)

Jens Frey Halling

Københavns Universitet

DENMARK

View details

Johan Onslev

Københavns Universitet

DENMARK

View details

Cathrine Nordgaard

Max Planck Institute for Infection Biology

GERMANY

View details

Nicoline Resen Andersen

Københavns Universitet

DENMARK

View details

Melanie Blasius

Københavns Universitet

DENMARK

View details

jørgen wojtaszewski

Københavns Universitet

DENMARK

View details

Janne Rasmus Hingst

Københavns Universitet

DENMARK

View details

Emeline Perrier-Groult

FRANCE

View details

Jonas Treebak

Københavns Universitet

DENMARK

View details

Thomas Jensen

Københavns Universitet

DENMARK

View details

Jesper Bratz Birk

Københavns Universitet

DENMARK

View details

Jonas Kristensen

Københavns Universitet

DENMARK

View details

Rasmus Kjøbsted

Københavns Universitet

DENMARK

View details

Cristian Pablo Pennisi

Aalborg University

DENMARK

View details

Anna Constance Vind

Københavns Universitet

DENMARK

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Nicolas O. Eskesen

Research Interests

Contact this professor

Articles (8)

The human AMPKγ3 R225W mutation negatively impacts site‐1 nucleotide binding and does not enhance basal AMPKγ3‐associated activity nor glycogen production in human or mouse skeletal muscle

Energy sensor AMPKγ does not exist in isolation from AMPKα or interact with PPP6C in muscle and liver from humans and mice.

TBC1D4-S711 controls skeletal muscle insulin sensitization after exercise and contraction

AMPKγ3 Controls Muscle Glucose Uptake in Recovery From Exercise to Recapture Energy Stores.

ZAKβ is activated by cellular compression and mediates contraction‐induced <scp>MAP</scp> kinase signaling in skeletal muscle

Direct small molecule ADaM-site AMPK activators reveal an AMPKγ3-independent mechanism for blood glucose lowering

Measurement of insulin- and contraction-stimulated glucose uptake in isolated and incubated mature skeletal muscle from mice

Inducible deletion of skeletal muscle AMPKα 1 reveals that AMPK is required for nucleotide balance but dispensable for muscle glucose uptake and fat oxidation during exercise

Collaborators (15)

Jens Frey Halling

Johan Onslev

Cathrine Nordgaard

Nicoline Resen Andersen

Melanie Blasius

jørgen wojtaszewski

Janne Rasmus Hingst

Emeline Perrier-Groult

Jonas Treebak

Thomas Jensen

Jesper Bratz Birk

Jonas Kristensen

Rasmus Kjøbsted

Cristian Pablo Pennisi

Anna Constance Vind

Make your next move.