Nikhlesh Singh

Associate Professor

Wayne State University School of Medicine

United States

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Dr. Nikhlesh Singh is an Associate Professor at Wayne State University School of Medicine in the United States. His research focuses on retinal diseases, with recent publications exploring topics such as the roles of IL-33 in ocular infections, the impact of fatty acids on age-related macular degeneration, and the application of nanotechnology in disease diagnosis and therapy. His work also investigates key regulatory mechanisms in endothelial cell activation and pathological neovascularization.

Articles (15)

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2025

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2025

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2025

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2024

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2024

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2023

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2023

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2023

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2022

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2022

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2022

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2021

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2021

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2021

Collaborators (2)

Linda D. Hazlett

Wayne State University School of Medicine

UNITED STATES

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Ashok Kumar

Asst. Prof

Wayne State University School of Medicine

UNITED STATES

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Nikhlesh Singh

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Articles (15)

A Disintegrin and Metalloproteinase 10 Regulates Ephrin B2–Mediated Endothelial Cell Sprouting and Ischemic Retinopathy

Implications of Fatty Acids for Age-Related Macular Degeneration: Evidence and Recommendations

Proteomic profiling reveals immunomodulatory role of IL-33 in ocular bacterial and fungal infections.

A neutrophil elastase-generated mature form of IL-33 is a potent regulator of endothelial cell activation and proliferative retinopathy

Nanotechnology in retinal diseases: From disease diagnosis to therapeutic applications

Vascular cell-adhesion molecule 1 (VCAM-1) regulates JunB-mediated IL-8/CXCL1 expression and pathological neovascularization

Long Non-Coding RNAs and Proliferative Retinal Diseases

IL-33 via PKCμ/PRKD1 Mediated α-Catenin Phosphorylation Regulates Endothelial Cell-Barrier Integrity and Ischemia-Induced Vascular Leakage

Role of G-Proteins and GPCRs in Cardiovascular Pathologies

IL-33 enhances Jagged1 mediated NOTCH1 intracellular domain (NICD) deubiquitination and pathological angiogenesis in proliferative retinopathy

Corrigendum to “ROS via BTK-p300-STAT1-PPARγ signaling activation mediates cholesterol crystals-induced CD36 expression and foam cell formation” [Redox Biol. 11 (2017) 350–364/513] (Redox Biology (2017) 11 (350–364), (S2213231716302725), (10.1016/j.redox.2016.12.005))

Erratum: Heterodimers of the transcriptional factors NFATc3 and FosB mediate tissue factor expression for 15(S)-hydroxyeicosatetraenoic acid-induced monocyte trafficking (Journal of Biological Chemistry DOI: 10.1074/jbc.M117.804344)

The Role of Inflammation in Retinal Neurodegeneration and Degenerative Diseases

Correction: Thrombin-Par1 signaling axis disrupts COP9 signalosome subunit 3-mediated ABCA1 stabilization in inducing foam cell formation and atherogenesis (Cell Death & Differentiation, (2021), 28, 2, (780-798), 10.1038/s41418-020-00623-9)

Nano-Biomaterials for Retinal Regeneration

Collaborators (2)

Linda D. Hazlett

Ashok Kumar

Make your next move.

Nikhlesh Singh

Research Interests

Explore related searches

Contact this professor

About

Articles (15)

A Disintegrin and Metalloproteinase 10 Regulates Ephrin B2–Mediated Endothelial Cell Sprouting and Ischemic Retinopathy

Implications of Fatty Acids for Age-Related Macular Degeneration: Evidence and Recommendations

Proteomic profiling reveals immunomodulatory role of IL-33 in ocular bacterial and fungal infections.

A neutrophil elastase-generated mature form of IL-33 is a potent regulator of endothelial cell activation and proliferative retinopathy

Nanotechnology in retinal diseases: From disease diagnosis to therapeutic applications

Vascular cell-adhesion molecule 1 (VCAM-1) regulates JunB-mediated IL-8/CXCL1 expression and pathological neovascularization

Long Non-Coding RNAs and Proliferative Retinal Diseases

IL-33 via PKCμ/PRKD1 Mediated α-Catenin Phosphorylation Regulates Endothelial Cell-Barrier Integrity and Ischemia-Induced Vascular Leakage

Role of G-Proteins and GPCRs in Cardiovascular Pathologies

IL-33 enhances Jagged1 mediated NOTCH1 intracellular domain (NICD) deubiquitination and pathological angiogenesis in proliferative retinopathy

Corrigendum to “ROS via BTK-p300-STAT1-PPARγ signaling activation mediates cholesterol crystals-induced CD36 expression and foam cell formation” [Redox Biol. 11 (2017) 350–364/513] (Redox Biology (2017) 11 (350–364), (S2213231716302725), (10.1016/j.redox.2016.12.005))

Erratum: Heterodimers of the transcriptional factors NFATc3 and FosB mediate tissue factor expression for 15(S)-hydroxyeicosatetraenoic acid-induced monocyte trafficking (Journal of Biological Chemistry DOI: 10.1074/jbc.M117.804344)

The Role of Inflammation in Retinal Neurodegeneration and Degenerative Diseases

Correction: Thrombin-Par1 signaling axis disrupts COP9 signalosome subunit 3-mediated ABCA1 stabilization in inducing foam cell formation and atherogenesis (Cell Death &amp; Differentiation, (2021), 28, 2, (780-798), 10.1038/s41418-020-00623-9)

Nano-Biomaterials for Retinal Regeneration

Collaborators (2)

Linda D. Hazlett

Ashok Kumar

Make your next move.

Correction: Thrombin-Par1 signaling axis disrupts COP9 signalosome subunit 3-mediated ABCA1 stabilization in inducing foam cell formation and atherogenesis (Cell Death & Differentiation, (2021), 28, 2, (780-798), 10.1038/s41418-020-00623-9)