Nikolaos G. Frangogiannis

Albert Einstein College of Medicine

United States

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Articles (21)

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2025

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2021

Collaborators (3)

Anis Hanna

Research Assistant Professor

Albert Einstein College of Medicine

UNITED STATES

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Akihiko Kubota

Albert Einstein College of Medicine

UNITED STATES

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Deyou Zheng

Assistant/Associate Professor/Professor

Albert Einstein College of Medicine

UNITED STATES

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Nikolaos G. Frangogiannis

Research Interests

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Articles (21)

Fibroblast-Specific Loss of TGF-β Signaling Mediates Lipomatous Metaplasia in the Infarcted Heart

Pericytes in tissue fibrosis

Cardiomyocytes Regulate Inflammation in the Failing Heart

Targeting macrophage–fibroblast interactions in the failing heart

Fibroblast-specific TGF-β signaling mediates cardiac dysfunction, fibrosis, and hypertrophy in obese diabetic mice

The fate and role of the pericytes in myocardial diseases

Targeting metabolically activated fibroblasts in the failing heart

Repair of the Infarcted Heart: Cellular Effectors, Molecular Mechanisms and Therapeutic Opportunities

Response by Frangogiannis to Letter Regarding Article, “Cardiac Pericytes Acquire a Fibrogenic Phenotype and Contribute to Vascular Maturation After Myocardial Infarction”

Diabetes Induces Cardiac Fibroblast Activation, Promoting a Matrix‐Preserving Nonmyofibroblast Phenotype, Without Stimulating Pericyte to Fibroblast Conversion

Cardiac Pericytes Acquire a Fibrogenic Phenotype and Contribute to Vascular Maturation After Myocardial Infarction

Targeting galectin-3 in myocardial infarction: a unique opportunity for biomarker-guided therapy

Properties and Functions of Fibroblasts and Myofibroblasts in Myocardial Infarction

Smad7 effects on TGF-β and ErbB2 restrain myofibroblast activation and protect from postinfarction heart failure

Macrophages in myocardial infarction.

The role of endogenous Smad7 in regulating macrophage phenotype following myocardial infarction

Transforming growth factor-β in myocardial disease

Smad-dependent pathways in the infarcted and failing heart

Validation of Specific and Reliable Genetic Tools to Identify, Label, and Target Cardiac Pericytes in Mice

Cardiac fibrosis

Collagen denaturation in the infarcted myocardium involves temporally distinct effects of MT1-MMP-dependent proteolysis and mechanical tension

Collaborators (3)

Anis Hanna

Akihiko Kubota

Deyou Zheng

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