Nitin Karandikar

Professor and Chair/DEO

University of Iowa Healthcare

United States

Research Interests

Explore related searches

Professors in United States Professors in Immunology Immunology professors in United States

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

About

Dr. Nitin Karandikar is a Professor and Chair at the University of Iowa Health Care in the United States. His research primarily focuses on autoimmune diseases and the immunological mechanisms involved in conditions such as multiple sclerosis. Recent publications include studies on Th17-mediated autoimmunity, CD8+ T cell functionality, and the role of dendritic cells in autoimmune responses.

Articles (14)

Year:

2025

Year:

2025

Year:

2025

Year:

2024

Year:

2023

Year:

2023

Year:

2022

Year:

2022

Year:

2021

Year:

2021

Year:

2020

Year:

2020

Year:

2020

Year:

2020

Collaborators (6)

Sterling B. Ortega

UNITED STATES

View details

Alexander Boyden

University of Iowa Healthcare

UNITED STATES

View details

Enrico Radaelli

Associate Professor

University of Pennsylvania

UNITED STATES

View details

Jian Zhang

Professor of pathology and Endowed Chair in Immunology Research

Iowa State University

UNITED STATES

View details

Michael Tansey

Iowa State University

UNITED STATES

View details

Carol J Holman

University of Iowa Hospitals and Clinics

UNITED STATES

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Nitin Karandikar

Research Interests

Explore related searches

Contact this professor

About

Articles (14)

Correction: K27-linked RORγt ubiquitination by Nedd4 potentiates Th17-mediated autoimmunity.

Myelin-reactive CD8+ T cells influence conventional dendritic cell subsets towards a mature and regulatory phenotype in experimental autoimmune encephalomyelitis

K27-linked RORγt ubiquitination by Nedd4 potentiates Th17-mediated autoimmunity.

Disruption of IFNγ, GZMB, PRF1, or LYST Results in Reduced Suppressive Function in Human CD8+ T Cells

Proteolipid Protein–Induced Mouse Model of Multiple Sclerosis Requires B Cell–Mediated Antigen Presentation

IL-17 cytokines preferentially act on naïve CD4+ T cells with the IL-17AF heterodimer inducing the greatest functional changes

Perfusion with 10% neutral-buffered formalin is equivalent to 4% paraformaldehyde for histopathology and immunohistochemistry in a mouse model of experimental autoimmune encephalomyelitis

Hyperacute immune responses associate with immediate neuropathology and motor dysfunction in large vessel occlusions.

A Functionally Distinct CXCR3+/IFN-γ+/IL-10+ Subset Defines Disease-Suppressive Myelin-Specific CD8 T Cells

Utility of Flow Cytometry and Fluorescence In Situ Hybridization in Follow-up Monitoring of Plasma Cell Myeloma.

Altered expression of SIRPγ on the T-cells of relapsing remitting multiple sclerosis and type 1 diabetes patients could potentiate effector responses from T-cells

Human Commensal <i>Prevotella histicola</i> Ameliorates Disease as Effectively as Interferon-Beta in the Experimental Autoimmune Encephalomyelitis.

IL-12-Induced Immune Suppressive Deficit During CD8+ T-Cell Differentiation.

CD4 T cell-intrinsic role for the T helper 17 signature cytokine IL-17: Effector resistance to immune suppression

Collaborators (6)

Sterling B. Ortega

Alexander Boyden

Enrico Radaelli

Jian Zhang

Michael Tansey

Carol J Holman

Make your next move.