Noriyuki Masaki

University of California, San Diego

United States

Research Interests

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Articles (12)

Collaborators (3)

Takamitsu Inoue

International University of Health and Welfare

Jun Miyazaki

International University of Health and Welfare

YASUNORI TOME

University of the Ryukyus

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Noriyuki Masaki

Research Interests

Make your next move.

Explore related searches

Articles (12)

Collaborators (3)

Takamitsu Inoue

Jun Miyazaki

YASUNORI TOME

Make your next move.

Noriyuki Masaki

Research Interests

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Explore related searches

Articles (12)

Targeting Methionine Addiction of Osteosarcoma with Methionine Restriction to Overcome Drug Resistance: A New Paradigm for a Recalcitrant Disease

Methionine restriction of glioma does not induce MGMT and greatly improves temozolomide efficacy in an orthotopic nude-mouse model: A potential curable approach to a clinically-incurable disease

The First Mouse Model of Meckel's Diverticulum Carcinoma

Deletion of MTAP Highly Sensitizes Osteosarcoma Cells to Methionine Restriction With Recombinant Methioninase

Non-invasively Imageable Tibia-tumor-fragment Implantation Experimental-bone-metastasis Mouse Model of GFP-expressing Prostate Cancer

Oral-recombinant Methioninase Converts an Osteosarcoma from Methotrexate-resistant to -sensitive in a Patient-derived Orthotopic-xenograft (PDOX) Mouse Model

Stage IV Pancreatic Cancer Patient Treated With FOLFIRINOX Combined With Oral Methioninase: A Highly-Rare Case With Long-term Stable Disease

Clinical features of multiply recurrent retroperitoneal liposarcoma: A single-center experience

Osteosarcoma of the breast in a Patient Derived Orthotopic Xenograft (PDOX) mouse model is arrested by both cisplatinum and eribulin

Histone H3 lysine-trimethylation markers are decreased by recombinant methioninase and increased by methotrexate at concentrations which inhibit methionine-addicted osteosarcoma cell proliferation

Over-methylation of Histone H3 Lysines Is a Common Molecular Change among the Three Major Types of Soft-tissue Sarcoma in Patient-derived Xenograft (PDX) Mouse Models

The combination of cisplatinum and doxorubicin regressed primary osteosarcoma of the breast in a PDOX mouse model

Collaborators (3)

Takamitsu Inoue

Jun Miyazaki

YASUNORI TOME

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