Octavio A. Gonzalez

The mechanisms through which oral commensal bacteria mitigates uncontrolled inflammatory responses of the oral mucosa remain unknown. Here, we show that representative oral bacterial species normally associated with oral health [ S. gordonii ( Sg ), V. parvula ( Vp ), A. naeslundii ( An ), C. sputigena ( Cs ), and N. mucosa ( Nm )] enhanced differential chemokine responses in oral epithelial cells (OECs), with some bacteria ( An, Vp, and Nm ) inducing higher chemokine levels (CXCL1, CXCL8) than others ( Sg, Cs ).