Paolo Gandellini

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Associate Professor

University of Milan

Italy

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About

Paolo Gandellini is an Associate Professor at the University of Milan, Italy. His research focuses on cancer biology, particularly in the areas of G-quadruplex ligands, interferon signaling in bladder cancer, and the role of lncRNAs in pediatric neurological diseases. He has contributed to understanding cellular adaptations and post-transcriptional control mechanisms in various cancers, as highlighted by his recent publications.

Recent Grants

Grant: Close

Dissecting the role of NFY noncoding-ome in human cancer

Open Date: 2021-01-01

Close Date: 2025-01-01

Grant: Close

Dissecting the role of unexplored miR-205 host gene as long non-coding RNA in prostate basal cells: implications for cancer development

Open Date: 2016-01-01

Close Date: 2018-01-01

Grant: Close

Dissecting the role of unexplored miR-205 host gene as a basal cell-specific long non-coding RNA in prostate cancer development

Open Date: 2016-01-01

Close Date: 2018-01-01

Grant: Close

MicroRNA control of prostate cancer metastasis

Open Date: 2012-01-01

Close Date: 2014-01-01

Articles (13)

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2025

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2022

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2022

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2021

Collaborators (13)

Maurizio Colecchia

Professor

Università Vita Salute San Raffaele

ITALY

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paolo verderio

Fondazione IRCCS Istituto Nazionale dei Tumori

ITALY

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Riccardo Valdagni

Associate Professor

Nadia Zaffaroni

Head of Research Unit (Molecular Pharmacology)

Fondazione IRCCS Istituto Nazionale dei Tumori

ITALY

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Paolo Gandellini

Research Interests

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Recent Grants

Articles (13)

Cellular adaptations impact the biological activity of naphthalene diimide G-quadruplex ligands in ALT-positive osteosarcoma cells

LEADR, a p63 target, dampens interferon signalling in bladder cancer

Effective lowering of α-synuclein expression by targeting G-quadruplex structures within the SNCA gene.

Cleavage and polyadenylation machinery as a novel targetable vulnerability for human cancer

Repositioning of antiarrhythmics for prostate cancer treatment: a novel strategy to reprogram cancer-associated fibroblasts towards a tumor-suppressive phenotype.

Linc00941 fuels ribogenesis and protein synthesis by supporting robust cMYC translation in malignant pleural mesothelioma.

The pancancer overexpressed NFYC Antisense 1 controls cell cycle mitotic progression through in cis and in trans modes of action.

Post-transcriptional control drives Aurora kinase A expression in human cancers

Study of lncRNAs in Pediatric Neurological Diseases: Methods, Analysis of the State-of-Art and Possible Therapeutic Implications

Computational analysis of five neurodegenerative diseases reveals shared and specific genetic loci

Potential of the Stromal Matricellular Protein Periostin as a Biomarker to Improve Risk Assessment in Prostate Cancer

Navigating the Multiverse of Antisense RNAs: The Transcription- and RNA-Dependent Dimension

miR-1227 Targets SEC23A to Regulate the Shedding of Large Extracellular Vesicles

Collaborators (13)

Maurizio Colecchia

paolo verderio

Riccardo Valdagni

Federica Rey

Stephana Carelli

Filippo Doria

Valentina Doldi

Giovanni Centonze

Cristina Marenghi

Raffaella Zamponi

Silva Ljevar

jlenia guarnerio

Nadia Zaffaroni

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