Pedro Vale

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University of Edinburgh

United Kingdom

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Recent Grants

Grant: Close

What makes a super-spreader? The genetic architecture and evolutionary constraints on host heterogeneity in pathogen transmission

Open Date: 2022-11-01

Close Date: 2023-07-30

Grant: Close

Mitochondrial genetic effects on innate immunity in Drosophila

Open Date: 2018-04-01

Close Date:

Grant: Close

Evolution of virulence in immune-compromised hosts and the adaptation of emerging viruses

Open Date: 2017-07-01

Close Date: 2019-06-30

Grant: Close

Testing the evolutionary basis of the longevity response to dietary restriction

Open Date: 2017-04-01

Close Date:

Grant: Close

The causes and consequences of individual-level variation in disease tolerance

Open Date: 2015-01-01

Close Date: 2019-12-01

Articles (19)

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2020

Collaborators (9)

Hideharu Numata

特定教授 / Program-Specific Professor

Kyoto University

JAPAN

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Tiina Susanna Salminen

Tampere University

FINLAND

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Laura Vesala

University of Tampere

FINLAND

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Yuliya Basikhina

University of Tampere

FINLAND

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Fergal Waldron

University of Aberdeen

UNITED KINGDOM

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Per T Smiseth

University of Edinburgh

UNITED KINGDOM

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Andrew MacIntosh

Associate Professor

JAPAN

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Tea Tuomela

Tampere University

FINLAND

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Kenji Tomioka

Professor

Okayama University

JAPAN

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Pedro Vale

Research Interests

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Recent Grants

Articles (19)

Key questions for future research in Physiological Entomology

The ghost of infections past: Accounting for heterogeneity in individual infection history improves accuracy in epidemic forecasting

Mitochondrial background can explain variable costs of immune deployment

Negative immune regulation contributes to disease tolerance in Drosophila melanogaster

IMD-mediated innate immune priming increases Drosophila survival and reduces pathogen transmission

RETRACTED AND REPLACED: Mitochondrial background can explain variable costs of immune deployment

Mitochondrial perturbation in immune cells enhances cell-mediated innate immunity in Drosophila

A naturally occurring mitochondrial genome variant confers broad protection from infection in Drosophila

Genetic Variation in Trophic Avoidance Behaviour Shows Fruit Flies are Generally Attracted to Bacterial Substrates

Duox and Jak/Stat signalling influence disease tolerance in Drosophila during Pseudomonas entomophila infection

Intraspecific genetic variation in host vigour, viral load and disease tolerance during Drosophila C virus infection

Ageing leads to reduced specificity of antimicrobial peptide responses in Drosophila melanogaster

Variation in mitochondrial DNA affects locomotor activity and sleep in Drosophila melanogaster

Mechanisms of damage prevention, signalling and repair impact disease tolerance

Host genetics and pathogen species modulate infection-induced changes in social aggregation behaviour

Carry on caring: infected females maintain their parental care despite high mortality

Testing evolutionary explanations for the lifespan benefit of dietary restriction in fruit flies ( Drosophila melanogaster )

Dissecting genetic and sex-specific sources of host heterogeneity in pathogen shedding and spread

Genotype and sex-based host variation in behaviour and susceptibility drives population disease dynamics

Collaborators (9)

Hideharu Numata

Tiina Susanna Salminen

Laura Vesala

Yuliya Basikhina

Fergal Waldron

Per T Smiseth

Andrew MacIntosh

Tea Tuomela

Kenji Tomioka

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