Petr Cejka

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Institute for Research in Biomedicine

Switzerland

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Recent Grants

Grant: Open

BRCA INSIGHTS

Open Date: 2022-04-01

Close Date: 2027-03-01

Grant: Close

First steps in DNA double-strand break repair: underlying mechanisms and regulation

Open Date: 2021-10-01

Close Date: 2025-09-01

Grant: Close

HRMECH

Open Date: 2016-09-01

Close Date: 2022-03-01

Grant: Close

Repair of Damaged DNA by Homologous recombination and Related Pathways

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2020

Collaborators (7)

Renata Tisi

University of Milano-Bicocca

ITALY

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Xiaojing Mu

ETH Zürich

SWITZERLAND

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Antony W Oliver

University of Sussex

UNITED KINGDOM

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Bertrand Llorente

FRANCE

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Miki Shinohara

Professor

Kindai University

JAPAN

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Thomas Clarke

Assistant Professor

Boston University

UNITED STATES

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Virginie Ropars

Researcher

CEA Saclay

FRANCE

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Petr Cejka

Research Interests

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Recent Grants

Articles (20)

ZNF280A links DNA double-strand break repair to human 22q11.2 distal deletion syndrome

Tumor-associated neutrophil precursors impair homologous DNA repair and promote sensitivity to PARP inhibition.

EXO1 promotes the meiotic MLH1-MLH3 endonuclease through conserved interactions with MLH1, MSH4 and DNA.

MRN-CtIP, EXO1, and DNA2-WRN/BLM act bidirectionally to process DNA gaps in PARPi-treated cells without strand cleavage.

Comprehensive interrogation of synthetic lethality in the DNA damage response.

The PIN1-p38-CtIP signalling axis protects stalled replication forks from deleterious degradation.

Mechanisms and regulation of DNA end resection in the maintenance of genome stability.

Sae2 controls Mre11 endo- and exonuclease activities by different mechanisms

Mechanism of BRCA1–BARD1 function in DNA end resection and DNA protection

Exploring the removal of Spo11 and topoisomerases from DNA breaks in S. cerevisiae by human Tyrosyl DNA Phosphodiesterase 2.

Crossover or non-crossover outcomes: tailored processing of homologous recombination intermediates.

Inhibition of MRN activity by a telomere protein motif.

Molecular basis of the dual role of the Mlh1-Mlh3 endonuclease in MMR and in meiotic crossover formation.

The Pif1 helicase is actively inhibited during meiotic recombination which restrains gene conversion tract length.

Sae2 and Rif2 regulate MRX endonuclease activity at DNA double-strand breaks in opposite manners.

Single-molecule studies illuminate the function of RAD51 paralogs.

MRE11-RAD50-NBS1 Complex Is Sufficient to Promote Transcription by RNA Polymerase II at Double-Strand Breaks by Melting DNA Ends.

A Disease-Causing Single Amino Acid Deletion in the Coiled-Coil Domain of RAD50 Impairs MRE11 Complex Functions in Yeast and Humans.

Exo1 recruits Cdc5 polo kinase to MutLγ to ensure efficient meiotic crossover formation

MCM8IP activates the MCM8-9 helicase to promote DNA synthesis and homologous recombination upon DNA damage

Collaborators (7)

Renata Tisi

Xiaojing Mu

Antony W Oliver

Bertrand Llorente

Miki Shinohara

Thomas Clarke

Virginie Ropars

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