Petronel Tuluc

University of Innsbruck

Austria

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Articles (17)

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2025

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2025

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2025

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2024

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2023

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2022

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2021

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2021

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2021

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2021

Collaborators (9)

Marta Campiglio

AUSTRIA

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Nadine J Ortner

AUSTRIA

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Andrea Marcantoni

University of Turin

ITALY

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Jörg Striessnig

University of Innsbruck

AUSTRIA

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X Shawn Liu

Columbia University

UNITED STATES

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Bernhard Flucher

Univ.-Prof.

Medical University of Innsbruck

AUSTRIA

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Stefanie M. Geisler

University of Innsbruck

AUSTRIA

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Sabrin Haddad

Karl Landsteiner University of Health Sciences

AUSTRIA

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Emilio Carbone

Full professor, Deputy Director of Research

University of Turin

ITALY

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Petronel Tuluc

Research Interests

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Articles (17)

Voltage-sensor gating charge interactions bimodally regulate voltage dependence and kinetics of calcium channel activation

Two pairs of CACNA1I (CaV3.3) variants with opposite effects on channel function cause neurodevelopmental disorders of varying severity.

STAC3 binding to CaV1.1 II-III loop is nonessential but critically supports skeletal muscle excitation-contraction coupling

Novel protocol for multiple-dose oral administration of the L-type Ca <sup>2+</sup> channel blocker isradipine in mice: A dose-finding pharmacokinetic study

Deletion of the α2δ‐1 calcium channel subunit increases excitability of mouse chromaffin cells

CaV1.1 voltage-sensing domain III exclusively controls skeletal muscle excitation-contraction coupling

Molecular mechanism responsible for sex differences in electrical activity of mouse pancreatic β-cells

Excitation-secretion coupling in chromaffin cells of the adrenal gland: Where does calcium come from?

A genetically encoded actuator boosts L-type calcium channel function in diverse physiological settings

A biallelic mutation in <scp>CACNA2D2</scp> associated with developmental and epileptic encephalopathy affects calcium channel‐dependent as well as synaptic functions of α<sub>2</sub>δ‐2

The human channel gating-modifying A749G CACNA1D (Cav1.3) variant induces a neurodevelopmental syndrome-like phenotype in mice

STAC3 determines the slow activation kinetics of Ca<sub>V</sub>1.1 currents and inhibits its voltage‐dependent inactivation

Calcium current modulation by the γ1 subunit depends on alternative splicing of CaV1.1

Ion-pair interactions between voltage-sensing domain IV and pore domain I regulate CaV1.1 gating

Structural determinants of voltage-gating properties in calcium channels

Exciting leak: Na+ background current makes chromaffin cells burst

Stabilization of negative activation voltages of Cav1.3 L-Type Ca2+-channels by alternative splicing

Collaborators (9)

Marta Campiglio

Nadine J Ortner

Andrea Marcantoni

Jörg Striessnig

X Shawn Liu

Bernhard Flucher

Stefanie M. Geisler

Sabrin Haddad

Emilio Carbone

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