Prabhakara Nagareddy

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University of Oklahoma Health Sciences Center

United States

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About

Professor Prabhakara Nagareddy is a faculty member at the University of Oklahoma Health Sciences Center, USA. His research primarily focuses on adipocyte metabolism, macrophage heterogeneity in atherosclerosis, and the impact of obesity on inflammation and metabolic conditions. Recent publications highlight his work on therapeutic implications for myocardial infarction and the role of microbiomes in visceral adipose tissue inflammation.

Recent Grants

Grant: Close

Harnessing the potential of an endogenous anti-inflammatory mechanism to mitigate cardiac inflammation and heart failure

Open Date: 2022-07-01

Close Date: 2025-06-30

Grant: Close

SYNDECAN-4 AS A MOLECULAR LINK BETWEEN ADIPOSE TISSUE AND AGING

Open Date: 2020-08-01

Close Date: 2022-05-01

Grant: Close

Thrombocytopoiesis in Diabetes: Role of Damage Associated Molecular Patterns

Open Date: 2018-04-23

Close Date: 2023-03-31

Grant: Close

Adipose tissue macrophages promote myeloid cell proliferation and monocytosis in obesity

Open Date: 2014-05-01

Close Date: 2014-07-31

Grant: Close

Adipose tissue macrophages promote myeloid cell proliferation and monocytosis in obesity

Open Date: 2014-05-01

Close Date: 2014-07-31

Articles (20)

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2025

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2020

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2020

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2020

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2020

Collaborators (6)

Chandrasekhar R. Kothapalli

Cleveland State University

UNITED STATES

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Nordin M.J. Hanssen

Amsterdam University Medical Centers

NETHERLANDS

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Andrew Murphy

Associate Professor

Baker Heart and Diabetes Institute

AUSTRALIA

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Gregory Mullen

Research Assistant Professor

University of Oklahoma Health Sciences Center

UNITED STATES

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Bart Spaetgens

NETHERLANDS

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Albert DAHDAH

Karolinska Institutet

SWEDEN

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Prabhakara Nagareddy

Research Interests

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About

Recent Grants

Articles (20)

Activating nuclear receptor subfamily 2 group F member 2 in adipocyte stem cells rescues beige adipocyte metabolism impaired by excess early-life omega-6 fatty acids

Redefining Macrophage Heterogeneity in Atherosclerosis: A Focus on Possible Therapeutic Implications

Disulfiram Reduces Atherosclerosis and Enhances Efferocytosis, Autophagy, and Atheroprotective Gut Microbiota in Hyperlipidemic Mice

Impact of age and sex on myelopoiesis and inflammation during myocardial infarction

Key questions and gaps in understanding adipose tissue macrophages and early-life metabolic programming

Macrophage heterogeneity in myocardial infarction: Evolution and implications for diverse therapeutic approaches

Obesity-associated microbiomes instigate visceral adipose tissue inflammation by recruitment of distinct neutrophils

Administration of an LXR agonist promotes atherosclerotic lesion remodelling in murine inflammatory arthritis

Association between skeletal muscle mitochondrial dysfunction and insulin resistance in patients with rheumatoid arthritis: a case–control study

Correction: Syndecan-4 as a genetic determinant of the metabolic syndrome (Diabetology & Metabolic Syndrome, (2023), 15, 1, (156), 10.1186/s13098-023-01132-8)

Neutrophils in cardiovascular disease: warmongers, peacemakers, or both?

Immunological Insights into Cigarette Smoking-Induced Cardiovascular Disease Risk

Response by Nagareddy and Sreejit to Letter Regarding Article, “Retention of the NLRP3 Inflammasome-Primed Neutrophils in the Bone Marrow Is Essential for Myocardial Infarction–Induced Granulopoiesis”

Oxidative Stress in Neutrophils: Implications for Diabetic Cardiovascular Complications

Letter by Nagareddy and Sreejit Regarding Article, “Interleukin-1α Is a Central Regulator of Leukocyte-Endothelial Adhesion in Myocardial Infarction and in Chronic Kidney Disease”

DAMPening Mortality in COVID-19

NETosis Is Required for S100A8/A9-Induced Granulopoiesis After Myocardial Infarction

Emerging roles of neutrophil-borne S100A8/A9 in cardiovascular inflammation

Characterizing the Retinal Phenotype in the High-Fat Diet and Western Diet Mouse Models of Prediabetes

Neutrophil-Derived S100A8/A9 Amplify Granulopoiesis Following Myocardial Infarction

Collaborators (6)

Chandrasekhar R. Kothapalli

Nordin M.J. Hanssen

Andrew Murphy

Gregory Mullen

Bart Spaetgens

Albert DAHDAH

Make your next move.

Prabhakara Nagareddy

Research Interests

Explore related searches

Contact this professor

About

Recent Grants

Articles (20)

Activating nuclear receptor subfamily 2 group F member 2 in adipocyte stem cells rescues beige adipocyte metabolism impaired by excess early-life omega-6 fatty acids

Redefining Macrophage Heterogeneity in Atherosclerosis: A Focus on Possible Therapeutic Implications

Disulfiram Reduces Atherosclerosis and Enhances Efferocytosis, Autophagy, and Atheroprotective Gut Microbiota in Hyperlipidemic Mice

Impact of age and sex on myelopoiesis and inflammation during myocardial infarction

Key questions and gaps in understanding adipose tissue macrophages and early-life metabolic programming

Macrophage heterogeneity in myocardial infarction: Evolution and implications for diverse therapeutic approaches

Obesity-associated microbiomes instigate visceral adipose tissue inflammation by recruitment of distinct neutrophils

Administration of an LXR agonist promotes atherosclerotic lesion remodelling in murine inflammatory arthritis

Association between skeletal muscle mitochondrial dysfunction and insulin resistance in patients with rheumatoid arthritis: a case–control study

Correction: Syndecan-4 as a genetic determinant of the metabolic syndrome (Diabetology &amp; Metabolic Syndrome, (2023), 15, 1, (156), 10.1186/s13098-023-01132-8)

Neutrophils in cardiovascular disease: warmongers, peacemakers, or both?

Immunological Insights into Cigarette Smoking-Induced Cardiovascular Disease Risk

Response by Nagareddy and Sreejit to Letter Regarding Article, “Retention of the NLRP3 Inflammasome-Primed Neutrophils in the Bone Marrow Is Essential for Myocardial Infarction–Induced Granulopoiesis”

Oxidative Stress in Neutrophils: Implications for Diabetic Cardiovascular Complications

Letter by Nagareddy and Sreejit Regarding Article, “Interleukin-1α Is a Central Regulator of Leukocyte-Endothelial Adhesion in Myocardial Infarction and in Chronic Kidney Disease”

DAMPening Mortality in COVID-19

NETosis Is Required for S100A8/A9-Induced Granulopoiesis After Myocardial Infarction

Emerging roles of neutrophil-borne S100A8/A9 in cardiovascular inflammation

Characterizing the Retinal Phenotype in the High-Fat Diet and Western Diet Mouse Models of Prediabetes

Neutrophil-Derived S100A8/A9 Amplify Granulopoiesis Following Myocardial Infarction

Collaborators (6)

Chandrasekhar R. Kothapalli

Nordin M.J. Hanssen

Andrew Murphy

Gregory Mullen

Bart Spaetgens

Albert DAHDAH

Make your next move.

Correction: Syndecan-4 as a genetic determinant of the metabolic syndrome (Diabetology & Metabolic Syndrome, (2023), 15, 1, (156), 10.1186/s13098-023-01132-8)