Qingyi Ma

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Assistant Professor

Loma Linda University

United States

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About

Qingyi Ma is an Assistant Professor at Loma Linda University in the United States. His research areas focus on neonatal brain injury, neuroinflammation, and the role of microRNAs in neurological disorders, as reflected in his recent publications. Notable works include studies on the effects of microRNA-210 on mitochondrial metabolism and vascular injury in hypoxic-ischemic conditions, as well as investigations into cerebral blood flow assessment using advanced imaging techniques.

Recent Grants

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The protective effects of c-type natriuretic peptide in the neonatal brain

Open Date: 2019-05-15

Close Date: 2024-02-29

Grant: Close

Neuroprotection of miR-210 inhibition in neonatal stroke

Open Date: 2015-07-01

Close Date: 2017-06-30

Articles (14)

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2025

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2024

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2023

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2021

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2021

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2021

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2020

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2020

Collaborators (3)

Kassandra Kisler

University of Southern California

UNITED STATES

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Lubo Zhang

Director/Professor

Loma Linda University

UNITED STATES

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Guofang Shen

City of Hope

UNITED STATES

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Qingyi Ma

Research Interests

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About

Recent Grants

Articles (14)

MicroRNA210 Suppresses Mitochondrial Metabolism and Promotes Microglial Activation in Neonatal Hypoxic–Ischemic Brain Injury

<i>Atp13a5</i> Marker Reveals Pericyte Specification in the Mouse Central Nervous System

3D doppler ultrasound imaging of cerebral blood flow for assessment of neonatal hypoxic-ischemic brain injury in mice.

MicroRNA-210 Downregulates TET2 (Ten-Eleven Translocation Methylcytosine Dioxygenase 2) and Contributes to Neuroinflammation in Ischemic Stroke of Adult Mice.

MicroRNA-210 Downregulates TET2 (Ten-Eleven Translocation Methylcytosine Dioxygenase 2) and Contributes to Neuroinflammation in Ischemic Stroke of Adult Mice

Fetal hypoxia results in sex- and cell type-specific alterations in neonatal transcription in rat oligodendrocyte precursor cells, microglia, neurons, and oligodendrocytes

C-Type Natriuretic Peptide Ameliorates Vascular Injury and Improves Neurological Outcomes in Neonatal Hypoxic-Ischemic Brain Injury in Mice.

MicroRNA-210 downregulates TET2 and contributes to inflammatory response in neonatal hypoxic-ischemic brain injury

Mild traumatic brain injury induces microvascular injury and accelerates Alzheimer-like pathogenesis in mice.

Endothelial LRP1 protects against neurodegeneration by blocking cyclophilin A

C-Type Natriuretic Peptide Ameliorates Vascular Injury and Improves Neurological Outcomes in Neonatal Hypoxic-Ischemic Brain Injury in Mice

Mild traumatic brain injury induces microvascular injury and accelerates Alzheimer-like pathogenesis in mice

Antenatal Hypoxia Accelerates the Onset of Alzheimer's Disease Pathology in 5xFAD Mouse Model.

MicroRNAs in the Blood-Brain Barrier in Hypoxic-Ischemic Brain Injury.

Collaborators (3)

Kassandra Kisler

Lubo Zhang

Guofang Shen

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