Qingyun Liu

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Assistant Professor

University of North Carolina at Chapel Hill

United States

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About

Qingyun Liu is an Assistant Professor at the University of North Carolina at Chapel Hill, United States. His research primarily focuses on the evolutionary dynamics of Mycobacterium tuberculosis and related pathogens, exploring themes such as drug resistance, host-pathogen interactions, and genomic adaptations. Recent publications include insights on the ongoing evolution of PE/PPE genes and macrolide resistance in Mycobacterium abscessus, as well as advancements in genomic tools for understanding tuberculosis transmission and treatment outcomes.

Recent Grants

Grant: Close

Genetic basis of the excessive accumulation of triacylglycerol in M. tuberculosis modern Beijing sublineage

Open Date: 2018-01-01

Close Date: 2020-12-01

Grant: Close

Genomic history of tuberculosis epidemic in China

Open Date: 2017-09-01

Close Date: 2018-09-01

Articles (13)

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2025

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2025

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2024

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2024

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2024

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2022

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2022

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2022

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2021

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2021

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2021

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2021

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2021

Collaborators (2)

Xin Wang

Harvard T.H. Chan School of Public Health

UNITED STATES

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Sydney Stanley

University of Pennsylvania

UNITED STATES

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Qingyun Liu

Research Interests

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About

Recent Grants

Articles (13)

Ongoing evolution of PE/PPE genes in Mycobacterium tuberculosis associated with drug resistance and host immune response

Macrolide resistance in Mycobacterium abscessus: current insights and future perspectives

An additional proofreader contributes to DNA replication fidelity in mycobacteria

Genetically encoded transcriptional plasticity underlies stress adaptation in Mycobacterium tuberculosis

Paired analysis of host and pathogen genomes identifies determinants of human tuberculosis

Tuberculosis treatment failure associated with evolution of antibiotic resilience

Modern Beijing sublineage of Mycobacterium tuberculosis shift macrophage into a hyperinflammatory status

SAM-TB: A whole genome sequencing data analysis website for detection of Mycobacterium tuberculosis drug resistance and transmission

Population genomics provides insights into the evolution and adaptation to humans of the waterborne pathogen Mycobacterium kansasii

Local adaptation of Mycobacterium tuberculosis on the Tibetan Plateau

Mycobacterial fatty acid catabolism is repressed by FdmR to sustain lipogenesis and virulence

A population-based genomic epidemiological study of the source of tuberculosis infections in an emerging city: Shenzhen, China

Deep whole-genome sequencing reveals no evidence for heteroresistance influencing treatment outcomes among drug-susceptible tuberculosis patients

Collaborators (2)

Xin Wang

Sydney Stanley

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