Rajendra Karki

Assistant Professor

Seoul National University

South Korea

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About

Rajendra Karki is an Assistant Professor at Seoul National University in South Korea. His research focuses on molecular mechanisms related to cell death pathways, innate immunity, and their implications in metabolic and cardiovascular diseases. Recent articles highlight his work on apoptosis, PANoptosis, and the roles of various components in inflammation and tumorigenesis.

Articles (14)

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2024

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2024

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2024

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2024

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2023

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2023

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2022

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2022

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2022

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2022

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2022

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2022

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2022

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2021

Collaborators (2)

Joo-Hui Han

Assistant professor

SOUTH KOREA

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SangJoon Lee

Assistant Professor

Korea Advanced Institute of Science and Technology

SOUTH KOREA

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Rajendra Karki

Research Interests

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Articles (14)

DNA sensors in metabolic and cardiovascular diseases: Molecular mechanisms and therapeutic prospects

A comparative study of apoptosis, pyroptosis, necroptosis, and PANoptosis components in mouse and human cells

NINJ1 mediates inflammatory cell death, PANoptosis, and lethality during infection conditions and heat stress

Chungtaejeon (CTJ) inhibits adhesion and migration of VSMC through cytoskeletal remodeling pathway

Regulated cell death pathways and their roles in homeostasis, infection, inflammation, and tumorigenesis

ADAR1 and ZBP1 in innate immunity, cell death, and disease.

Single cell analysis of PANoptosome cell death complexes through an expansion microscopy method

Innate immunity, cytokine storm, and inflammatory cell death in COVID-19.

Pancancer transcriptomic profiling identifies key PANoptosis markers as therapeutic targets for oncology.

Molecular mechanism of RIPK1 and caspase-8 in homeostatic type I interferon production and regulation.

ZBP1-dependent inflammatory cell death, PANoptosis, and cytokine storm disrupt IFN therapeutic efficacy during coronavirus infection.

NLRC4 Deficiency Leads to Enhanced Phosphorylation of MLKL and Necroptosis.

The Transcription Factor IRF9 Promotes Colorectal Cancer via Modulating the IL-6/STAT3 Signaling Axis.

Caspase-6 promotes activation of the caspase-11-NLRP3 inflammasome during gram-negative bacterial infections.

Collaborators (2)

Joo-Hui Han

SangJoon Lee

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