Rebecca Sosa

Assistant Professor of Pathology, Assistant Director of UCLA Immunogenetics Center

University of California, Los Angeles

United States

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Rebecca Sosa is an Assistant Professor of Pathology and the Assistant Director of the UCLA Immunogenetics Center at the University of California, Los Angeles. Her research focuses on immunogenetics, specifically the mechanisms of antibody-mediated rejection in transplantation, chronic inflammatory responses, and macrophage polarization. Recent publications highlight her work on issues related to cardiac allograft vasculopathy, liver transplantation, and immune system interactions in these contexts.

Articles (9)

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2024

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2024

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2023

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2023

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2022

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2021

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2020

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2020

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2020

Collaborators (5)

Aaron Meyer

Assistant Professor

University of California, Los Angeles

UNITED STATES

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Jessica Nevarez-Mejia

University of California, Los Angeles

UNITED STATES

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Elaine F. Reed

University of California

UNITED STATES

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Nicole Valenzuela

Assistant Professor in Residence

UCLA Health

UNITED STATES

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Bomi Lee

Assistant Professor

Stanford University

UNITED STATES

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Rebecca Sosa

Research Interests

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Articles (9)

Spatial multiomics of arterial regions from cardiac allograft vasculopathy rejected grafts reveal novel insights into the pathogenesis of chronic antibody-mediated rejection

Human leukocyte antigen class I antibody-activated endothelium promotes CD206+ M2 macrophage polarization and MMP9 secretion through TLR4 signaling and P-selectin in a model of antibody-mediated rejection and allograft vasculopathy

Disulfide-HMGB1 signals through TLR4 and TLR9 to induce inflammatory macrophages capable of innate-adaptive crosstalk in human liver transplantation

Immune Features of Disparate Liver Transplant Outcomes in Female Hispanics With Nonalcoholic Steatohepatitis

Cross-Talk between HLA Class I and TLR4 Mediates P-Selectin Surface Expression and Monocyte Capture to Human Endothelial Cells

T‐Cell Immunoglobulin and Mucin Domain‐Containing Protein‐4 Is Critical for Kupffer Cell Homeostatic Function in the Activation and Resolution of Liver Ischemia Reperfusion Injury

Antibody-induced vascular inflammation skews infiltrating macrophages to a novel remodeling phenotype in a model of transplant rejection.

Distinct immune characteristics distinguish hereditary and idiopathic chronic pancreatitis

Disulfide High-Mobility Group Box 1 Drives Ischemia-Reperfusion Injury in Human Liver Transplantation.

Collaborators (5)

Aaron Meyer

Jessica Nevarez-Mejia

Elaine F. Reed

Nicole Valenzuela

Bomi Lee

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