Richard Benninger

Associate Professor

University of Colorado Anschutz Medical Campus

United States

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About

Richard Benninger is an Associate Professor at the University of Colorado Anschutz Medical Campus School of Medicine in the United States. His research focuses on pancreatic beta-cell dynamics, specifically exploring electrical signaling, calcium oscillations, and the role of glucokinase in islet physiology. He has contributed to understanding the connectivity of beta-cell subgroups and their implications for glucose homeostasis and diabetes prevention.

Articles (13)

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2025

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2025

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2025

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2024

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2024

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2024

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2023

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2022

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2022

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2022

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2021

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2021

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2020

Collaborators (5)

JaeAnn Dwulet

Technical University of Munich

GERMANY

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Yunus Ozekin

Assistant Research Professor

University of Colorado Anschutz Medical Campus

UNITED STATES

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Matthew J. Merrins

Associate Professor

University of Wisconsin-Madison

UNITED STATES

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Kristen Wells

Assistant Research Professor

University of Colorado Anschutz Medical Campus

UNITED STATES

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Guy A. Rutter

UNITED KINGDOM

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Richard Benninger

Research Interests

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Articles (13)

Glucokinase activity controls peripherally located subpopulations of β-cells that lead islet Ca2+ oscillations

Glucokinase activity controls peripherally located subpopulations of β-cells that lead islet Ca2+ oscillations

Resolving Spatiotemporal Electrical Signaling Within the Islet via CMOS Microelectrode Arrays

Exploring pancreatic beta-cell subgroups and their connectivity

Depolarization induces calcium-dependent BMP4 release from mouse embryonic palate mesenchymal cells

Publisher Correction: Exploring pancreatic beta-cell subgroups and their connectivity

β-cell intrinsic dynamics rather than gap junction structure dictates subpopulations in the islet functional network

Dynamic changes in β-cell [Ca2+] regulate NFAT activation, gene transcription, and islet gap junction communication

Going With the Flow: Pericyte-Regulated Islet Blood Flow Influences Glucose Homeostasis

Ultrasound Imaging of Pancreatic Perfusion Dynamics Predicts Therapeutic Prevention of Diabetes in Preclinical Models of Type 1 Diabetes

Detecting insulitis in type 1 diabetes with ultrasound phase-change contrast agents

Small subpopulations of β-cells do not drive islet oscillatory [Ca2+] dynamics via gap junction communication

Contrast-enhanced ultrasound with sub-micron sized contrast agents detects insulitis in mouse models of type1 diabetes

Collaborators (5)

JaeAnn Dwulet

Yunus Ozekin

Matthew J. Merrins

Kristen Wells

Guy A. Rutter

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