Robert Shanks

Associate Professor

University of Pittsburgh

United States

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About

Robert Shanks is an Associate Professor at the University of Pittsburgh in the United States. His research areas encompass ocular infections, antimicrobial responses, and the biology of microorganisms such as Serratia marcescens and Enterococcus faecalis . Recent publications include investigations into the effects of antibiotics on bacterial stress responses, antiviral agents against ocular adenoviruses, and the inflammatory responses mediated by virulence factors.

Articles (10)

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2021

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2021

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2021

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2021

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (3)

Paul Leu

University of Pittsburgh

UNITED STATES

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Daria Van Tyne

Assistant Professor

University of Pittsburgh

UNITED STATES

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Regis Kowalski

Executive Director - Professor

University of Pittsburgh

UNITED STATES

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Robert Shanks

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Articles (10)

Antibiotics Used in Empiric Treatment of Ocular Infections Trigger the Bacterial Rcs Stress Response System Independent of Antibiotic Susceptibility

Speciation and Antibiotic Susceptibilities of Coagulase Negative Staphylococci Isolated from Ocular Infections

Genomic and phenotypic diversity of Enterococcus faecalis isolated from endophthalmitis

Differential susceptibility of airway and ocular surface cell lines to FlhDC-mediated virulence factors PhlA and ShlA from <i>Serratia marcescens</i>

Filociclovir Is an Active Antiviral Agent against Ocular Adenovirus Isolates In Vitro and in the Ad5/NZW Rabbit Ocular Model

The Rcs stress response system regulator GumB modulates <i>Serratia marcescens</i> induced inflammation and bacterial proliferation in a rabbit keratitis model and cytotoxicity <i>in vitro</i>

Topical Astodrimer Sodium, a Non-Toxic Polyanionic Dendrimer, Demonstrates Antiviral Activity in an Experimental Ocular Adenovirus Infection Model

Transcription Factor EepR Is Required for <i>Serratia marcescens</i> Host Proinflammatory Response by Corneal Epithelial Cells

Superhemophobic and Anti-Virofouling Coating for Mechanically Durable and Wash-Stable Medical Textiles

Biologically active pigment and ShlA cytolysin of Serratia marcescens induce autophagy in a human ocular surface cell line

Collaborators (3)

Paul Leu

Daria Van Tyne

Regis Kowalski

Make your next move.

Robert Shanks

Research Interests

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Contact this professor

About

Articles (10)

Antibiotics Used in Empiric Treatment of Ocular Infections Trigger the Bacterial Rcs Stress Response System Independent of Antibiotic Susceptibility

Speciation and Antibiotic Susceptibilities of Coagulase Negative Staphylococci Isolated from Ocular Infections

Genomic and phenotypic diversity of Enterococcus faecalis isolated from endophthalmitis

Differential susceptibility of airway and ocular surface cell lines to FlhDC-mediated virulence factors PhlA and ShlA from &lt;i&gt;Serratia marcescens&lt;/i&gt;

Filociclovir Is an Active Antiviral Agent against Ocular Adenovirus Isolates In Vitro and in the Ad5/NZW Rabbit Ocular Model

The Rcs stress response system regulator GumB modulates &lt;i&gt;Serratia marcescens&lt;/i&gt; induced inflammation and bacterial proliferation in a rabbit keratitis model and cytotoxicity &lt;i&gt;in vitro&lt;/i&gt;

Topical Astodrimer Sodium, a Non-Toxic Polyanionic Dendrimer, Demonstrates Antiviral Activity in an Experimental Ocular Adenovirus Infection Model

Transcription Factor EepR Is Required for &lt;i&gt;Serratia marcescens&lt;/i&gt; Host Proinflammatory Response by Corneal Epithelial Cells

Superhemophobic and Anti-Virofouling Coating for Mechanically Durable and Wash-Stable Medical Textiles

Biologically active pigment and ShlA cytolysin of Serratia marcescens induce autophagy in a human ocular surface cell line

Collaborators (3)

Paul Leu

Daria Van Tyne

Regis Kowalski

Make your next move.

Differential susceptibility of airway and ocular surface cell lines to FlhDC-mediated virulence factors PhlA and ShlA from <i>Serratia marcescens</i>

The Rcs stress response system regulator GumB modulates <i>Serratia marcescens</i> induced inflammation and bacterial proliferation in a rabbit keratitis model and cytotoxicity <i>in vitro</i>

Transcription Factor EepR Is Required for <i>Serratia marcescens</i> Host Proinflammatory Response by Corneal Epithelial Cells