Robert Zimmermann

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Assoc. Prof.

Graz University of Technology

Austria

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Robert Zimmermann is an Associate Professor at the University of Graz, Austria. His research focuses on lipid metabolism, specifically the mechanisms of lipolysis and the roles of various enzymes in lipid regulation. Recent publications highlight his work on bis(monoacylglycero)phosphate synthesis, phospholipase A2 functions, and the effects of lipid signaling on autoimmunity and inflammation.

Recent Grants

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SFB Lipid Hyrolysis (PP07) Metabolism of the late endosomal/lysosomal lipid bis(monoacylglycero)phosphate (BMP)

Open Date: 2019-01-01

Close Date: 2023-01-01

Grant: Close

Atglistatin-Preclinical development of small molecule inhibitors targeting human adipose triglyceride lipase

Open Date: 2017-05-01

Close Date: 2021-05-01

Grant: Close

Molecular mechanisms mediating ZAG signaling

Open Date: 2016-05-01

Close Date: 2021-04-30

Grant: Close

Molecular mechanisms mediating ZAG signaling

Open Date: 2016-01-01

Close Date: 2019-01-01

Grant: Close

Functional characterization of alpha/beta hydrolase domain-containing 6

Open Date: 2014-01-01

Close Date: 2017-12-31

Articles (20)

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2022

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2021

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2020

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2020

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2020

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2020

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2020

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2020

Collaborators (11)

Herbert Strobl

Professor of Immunology, Head of Research Division; Chairman

Medical University of Graz

AUSTRIA

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Alessandra Riva

Technical University of Munich

GERMANY

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Hanna Engelke

Graz University of Technology

AUSTRIA

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Ulrich Stelzl

professor

Graz University of Technology

AUSTRIA

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Achim Lass

Assoc. Prof. Mag. Dr.

Graz University of Technology

AUSTRIA

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Oliver Domenig

Head of Laboratory

AUSTRIA

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Gabriele Schoiswohl

Medical University of Graz

AUSTRIA

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Dagmar Kratky

Professor of Biochemistry

Medical University of Graz

AUSTRIA

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Carina Wagner

University of Graz

AUSTRIA

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Christian Wadsack

Medical University of Graz

AUSTRIA

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Bieke Broux

Hasselt University

BELGIUM

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Robert Zimmermann

Research Interests

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Recent Grants

Articles (20)

Inhibition of ATGL alleviates MASH via impaired PPARα signalling that favours hydrophilic bile acid composition in mice

Mutational scanning pinpoints distinct binding sites of key ATGL regulators in lipolysis

Functionally overlapping intra- and extralysosomal pathways promote bis(monoacylglycero)phosphate synthesis in mammalian cells

Phospholipase A2 group IVD mediates the transacylation of glycerophospholipids and acylglycerols

Fatty acid desaturation by stearoyl-CoA desaturase-1 controls regulatory T cell differentiation and autoimmunity.

Liver X Receptor Activation Attenuates Oxysterol-Induced Inflammatory Responses in Fetoplacental Endothelial Cells

Impact of (intestinal) LAL deficiency on lipid metabolism and macrophage infiltration

Cooperative lipolytic control of neuronal triacylglycerol by spastic paraplegia-associated enzyme DDHD2 and ATGL

Inhibition of diacylglycerol lipase β modulates lipid and endocannabinoid levels in the ex vivo human placenta

Lipid droplets control mitogenic lipid mediator production in human cancer cells

KIAA1363 affects retinyl ester turnover in cultured murine and human hepatic stellate cells.

Residues of the minimal sequence of G0S2 collectively contribute to ATGL inhibition while C-and N-terminal extensions promote binding to ATGL.

Hepatocyte-specific deletion of adipose triglyceride lipase (adipose triglyceride lipase/patatin-like phospholipase domain containing 2) ameliorates dietary induced steatohepatitis in mice.

Optimized expression and purification of adipose triglyceride lipase improved hydrolytic and transacylation activities in vitro.

Lysosomal acid lipase is the major acid retinyl ester hydrolase in cultured human hepatic stellate cells but not essential for retinyl ester degradation

Structure-activity relationship studies for the development of inhibitors of murine adipose triglyceride lipase (ATGL).

Dipeptidyl peptidase 3 modulates the renin-angiotensin system in mice.

Dipeptidyl peptidase 3 modulates the renin–Angiotensin system in mice

Dysregulated expression of monoacylglycerol lipase is a marker for anti-diabetic drug metformin-targeted therapy to correct impaired neurogenesis and spatial memory in Alzheimer's disease.

Metabolic regulation of the lysosomal cofactor bis(monoacylglycero)phosphate in mice.

Collaborators (11)

Herbert Strobl

Alessandra Riva

Hanna Engelke

Ulrich Stelzl

Achim Lass

Oliver Domenig

Gabriele Schoiswohl

Dagmar Kratky

Carina Wagner

Christian Wadsack

Bieke Broux

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