roberta foresti

Professor of Biochemistry

Université Paris-Est Créteil Val de Marne

France

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Professor Roberta Foresti is a faculty member in the Department of Biochemistry at Université Paris-Est Créteil Val de Marne, France. Her research focuses on phytochemistry, mitochondrial dysfunction, and metabolic responses in various biological contexts, as reflected in her recent publications. Notable areas of inquiry include the role of glutaminase in Wnt signaling, the effects of carbon monoxide-releasing molecules in disease models, and phospholipase activity in chronic disease states.

Articles (18)

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2025

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2024

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2023

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2023

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2023

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2022

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2022

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2022

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2021

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2021

Collaborators (11)

Aurélie Barrail-Tran

Université Paris-Saclay

FRANCE

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Michael Rivard

Université Paris-Est Créteil Val de Marne

FRANCE

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LEEYAH ISSOP

Institut Mondor de recherche biomédicale

FRANCE

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Sophie Féréol

Associate professor in cell biomechanics, Teaching in Physics and Mechanics

Université Paris-Est Créteil Val de Marne

FRANCE

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Claudio Bucolo

Associate Professor

Università degli Studi di Napoli Federico II

ITALY

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Hiroaki Kitagishi

Associate Professor

Doshisha University

JAPAN

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Sylvie Dufour

FRANCE

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Benjamin Even

Assistant Professor

Université Paris-Est Créteil Val de Marne

FRANCE

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Kazuhiko Igarashi

Prof.

Tohoku University graduate school of medicine

JAPAN

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Falk Buettner

Professor

Medizinische Hochschule Hannover

GERMANY

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Vijith Vijayan

Stanford University

UNITED STATES

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roberta foresti

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Articles (18)

Origanum majorana Extracts: A Preliminary Comparative Study on Phytochemical Profiles and Bioactive Properties of Valuable Fraction and By-Product

Non-canonical nuclear function of glutaminase cooperates with Wnt signaling to drive EMT during neural crest development

α1D Adrenergic Receptor Antagonism Protects Against High Glucose-Induced Mitochondrial Dysfunction and Blood Retinal Barrier Breakdown in ARPE-19 Cells

An oral carbon monoxide–releasing molecule protects against acute hyperhemolysis in sickle cell disease

Distinct metabolic responses to heme in inflammatory human and mouse macrophages - Role of nitric oxide.

An orally active carbon monoxide-releasing molecule enhances beneficial gut microbial species to combat obesity in mice.

Development of carbon monoxide-releasing molecules conjugated to polysaccharides (glyco-CORMs) for delivering CO during obesity

A synthetic porphyrin as an effective dual antidote against carbon monoxide and cyanide poisoning

Glucose oxidation drives trunk neural crest cell development and fate

Secreted phospholipase A2 XIIA triggers a mitochondrial damage-induced senescence in chronic obstructive pulmonary disease fibroblasts.

Glucose oxidation drives trunk neural crest cell development and fate

Secreted phospholipase A2 XIIA triggers a mitochondrial damage-induced senescence in chronic obstructive pulmonary disease fibroblasts

Genetic BACH1 deficiency alters mitochondrial function and increases NLRP3 inflammasome activation in mouse macrophages

CORM-401, an orally active carbon monoxide-releasing molecule, increases body temperature by activating non-shivering thermogenesis in rats

Inhibition of Adipose Tissue Beiging by HIV Integrase Inhibitors, Dolutegravir and Bictegravir, Is Associated with Adipocyte Hypertrophy, Hypoxia, Elevated Fibrosis, and Insulin Resistance in Simian Adipose Tissue and Human Adipocytes.

TLR4 Signaling and Heme Oxygenase-1/Carbon Monoxide Pathway Crosstalk Induces Resiliency of Myeloma Plasma Cells to Bortezomib Treatment

Metformin alleviates stress-induced cellular senescence of aging human adipose stromal cells and the ensuing adipocyte dysfunction.

Adipose tissue senescence is mediated by increased ATP content after a short-term high-fat diet exposure.

Collaborators (11)

Aurélie Barrail-Tran

Michael Rivard

LEEYAH ISSOP

Sophie Féréol

Claudio Bucolo

Hiroaki Kitagishi

Sylvie Dufour

Benjamin Even

Kazuhiko Igarashi

Falk Buettner

Vijith Vijayan

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