Rosanna Parlato

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Ruprecht-Karls-Universität Heidelberg Medizinische Fakultät Mannheim

Germany

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Recent Grants

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Role of primary cilia in striatal neurons for their vulnerability to degeneration in Huntington's disease

Open Date: 2018-01-01

Close Date: 2019-01-01

Grant: Close

The association of nucleolar activity with mutant huntingtin protein species in HD

Open Date: 2016-01-01

Close Date: 2017-01-01

Grant: Close

Role of nucleolar stress in the progression of neurodegeneration in models of Huntington´s disease.

Open Date: 2014-01-01

Close Date: 2018-12-31

Grant: Close

Screening of p53 targets in neurons under nucleolar stress leading to neurodegeneration

Open Date: 2011-01-01

Close Date: 2012-01-01

Articles (11)

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2024

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2023

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2023

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2022

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2022

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2022

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2022

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2021

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2021

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2020

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2020

Collaborators (11)

Kerry L. Tucker

UNITED STATES

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Julia Dyckow

Ruprecht-Karls-Universität Heidelberg Medizinische Fakultät Mannheim

GERMANY

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Deniz Yilmazer-Hanke

Professor, Section Head

Universität Ulm

GERMANY

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Philipp Koch

GERMANY

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Irena Nalepa

Professor, Ph.D., Head of Department

Maj Institute of Pharmacology Polish Academy of Sciences

POLAND

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Jie Jiang

Emory University

UNITED STATES

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Devesh Pant

Emory University

UNITED STATES

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Lu Rao

Research Assistant Professor

Albert Einstein College of Medicine

UNITED STATES

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JANANI PARAMESWARAN

Emory University

UNITED STATES

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Andreas Neueder

Professor

University Medical Center Hamburg-Eppendorf

GERMANY

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Carine Legrand

FRANCE

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Rosanna Parlato

Research Interests

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Recent Grants

Articles (11)

A TBK1 variant causes autophagolysosomal and motoneuron pathology without neuroinflammation in mice.

Queuosine‐tRNA promotes sex‐dependent learning and memory formation by maintaining codon‐biased translation elongation speed

Deep learning-based image analysis identifies a DAT-negative subpopulation of dopaminergic neurons in the lateral Substantia nigra.

ALS-linked KIF5A ΔExon27 mutant causes neuronal toxicity through gain-of-function.

Genetic lesions of the noradrenergic system trigger induction of oxidative stress and inflammation in the ventral midbrain

Oxidative Stress in Neurodegenerative Diseases

Treatment with THI, an inhibitor of sphingosine-1-phosphate lyase, modulates glycosphingolipid metabolism and results therapeutically effective in experimental models of Huntington's disease.

Targeted Ablation of Primary Cilia in Differentiated Dopaminergic Neurons Reduces Striatal Dopamine and Responsiveness to Metabolic Stress

Nucleolar stress controls mutant Huntington toxicity and monitors Huntington's disease progression.

Structural Fuzziness of the RNA-Organizing Protein SERF Determines a Toxic Gain-of-interaction

Nucleolar stress induces a senescence-like phenotype in smooth muscle cells and promotes development of vascular degeneration.

Collaborators (11)

Kerry L. Tucker

Julia Dyckow

Deniz Yilmazer-Hanke

Philipp Koch

Irena Nalepa

Jie Jiang

Devesh Pant

Lu Rao

JANANI PARAMESWARAN

Andreas Neueder

Carine Legrand

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