Ruslan Rafikov

Associate professor

University of Arizona

United States

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Dr. Ruslan Rafikov is an Associate Professor at the University of Arizona, USA. His research primarily focuses on pulmonary hypertension, mitochondrial dysfunction, and the role of cytokines in vascular health. Recent publications include studies on ARDS mortality predictive modeling, the impact of hemolysis on pulmonary hypertension, and the complexities of single-cell omics in cardiovascular diseases.

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2020

Collaborators (2)

Nancy Casanova

Research Associate Professor

University of Florida

UNITED STATES

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JIN SONG

Associate Research Professor

University of Florida

UNITED STATES

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Ruslan Rafikov

Research Interests

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About

Articles (20)

Mitochondrial dysfunction–induced hemolysis: a precursor to vascular leakage and pulmonary hypertension

Predictive modeling of ARDS mortality integrating biomarker/cytokine, clinical and metabolomic data

Deciphering the Complexities of Pulmonary Hypertension: The Emergent Role of Single-Cell Omics

Circulating free heme induces cytokine storm and pulmonary hypertension through the MKK3/p38 axis

eNAMPT neutralization reduces preclinical ARDS severity via rectified NFkB and Akt/mTORC2 signaling

Cytokine profiling in pulmonary arterial hypertension: the role of redox homeostasis and sex

Redox-dependent loss of flavin by mitochondria complex I is different in brain and heart

Endothelial eNAMPT drives EndMT and preclinical PH: rescue by an eNAMPT‐neutralizing mAb

Glucose-6-phosphate dehydrogenase deficiency contributes to metabolic abnormality and pulmonary hypertension

Glucose-6-phosphate dehydrogenase deficiency contributes to metabolic abnormality and pulmonary hypertension

Single Mutation in the NFU1 Gene Metabolically Reprograms Pulmonary Artery Smooth Muscle Cells

Necrosis-Released HMGB1 (High Mobility Group Box 1) in the Progressive Pulmonary Arterial Hypertension Associated With Male Sex

Complex III Inhibition-Induced Pulmonary Hypertension Affects the Mitochondrial Proteomic Landscape

Heme induces rapid endothelial barrier dysfunction via the MKK3/p38MAPK axis

Inhibition of Anaplerosis Attenuated Vascular Proliferation in Pulmonary Arterial Hypertension

Rats with a Human Mutation of NFU1 Develop Pulmonary Hypertension

Antioxidant-Conjugated Peptide Attenuated Metabolic Reprogramming in Pulmonary Hypertension.

Pulmonary Arterial Hypertension Induces a Distinct Signature of Circulating Metabolites.

Early progression of pulmonary hypertension in the monocrotaline model in males is associated with increased lung permeability

Sex-specific stress response and HMGB1 release in pulmonary endothelial cells

Collaborators (2)

Nancy Casanova

JIN SONG

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