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Sameer Bajikar

Assistant Professor

University of Virginia

United States

Research Interests

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Articles (5)

Year:

2026

Year:

2024

Year:

2024

Year:

2023

Year:

2023

Collaborators (7)

Sukru Aras

Baylor College of Medicine

UNITED STATES

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Tadahiro Mitani

Jichi Medical University

JAPAN

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Li Wang

Assistant Professor

Stanford University

UNITED STATES

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Davut Pehlivan

Baylor College of Medicine

UNITED STATES

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Jian Zhou

Assistant Professor

Emory University

UNITED STATES

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Hu Chen

Assistant Professor

Baylor College of Medicine

UNITED STATES

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Huda Y. Zoghbi

Director

Baylor College of Medicine

UNITED STATES

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Sameer Bajikar

Research Interests

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Articles (5)

Modulating alternative splicing of <i>MECP2</i> is a potential therapeutic strategy for Rett syndrome.

Structural variant allelic heterogeneity in MECP2 duplication syndrome provides insight into clinical severity and variability of disease expression

Modeling antisense oligonucleotide therapy in MECP2 duplication syndrome human iPSC-derived neurons reveals gene expression programs responsive to MeCP2 levels.

MeCP2 regulates Gdf11, a dosage-sensitive gene critical for neurological function

A novel pathogenic mutation of MeCP2 impairs chromatin association independent of protein levels

Collaborators (7)

Sukru Aras

Tadahiro Mitani

Li Wang

Davut Pehlivan

Jian Zhou

Hu Chen

Huda Y. Zoghbi

Make your next move.

Sameer Bajikar

Research Interests

Make your next move.

Explore related searches

Articles (5)

Modulating alternative splicing of &lt;i&gt;MECP2&lt;/i&gt; is a potential therapeutic strategy for Rett syndrome.

Structural variant allelic heterogeneity in MECP2 duplication syndrome provides insight into clinical severity and variability of disease expression

Modeling antisense oligonucleotide therapy in MECP2 duplication syndrome human iPSC-derived neurons reveals gene expression programs responsive to MeCP2 levels.

MeCP2 regulates Gdf11, a dosage-sensitive gene critical for neurological function

A novel pathogenic mutation of MeCP2 impairs chromatin association independent of protein levels

Collaborators (7)

Sukru Aras

Tadahiro Mitani

Li Wang

Davut Pehlivan

Jian Zhou

Hu Chen

Huda Y. Zoghbi

Make your next move.

Modulating alternative splicing of <i>MECP2</i> is a potential therapeutic strategy for Rett syndrome.