Samuel Mok

Professor

The University of Texas MD Anderson Cancer Center

United States

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Professor Samuel Mok is a faculty member at The University of Texas MD Anderson Cancer Center in the United States. His research focuses on molecular and metabolic profiling of tumor microenvironments, with recent publications exploring the immune microenvironment, ligand-receptor interactions in ovarian cancer, and cellular signaling in various cancer types. He is dedicated to identifying prognostic biomarkers and understanding the metabolic reprogramming of cancer cells.

Articles (11)

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2024

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2023

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2022

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2021

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2021

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2021

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2021

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2020

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2020

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2020

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2020

Collaborators (8)

Austin Miller

Asst Professor of Oncology, Biostatistics and Bioinformatics

Roswell Park Comprehensive Cancer Center

UNITED STATES

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Paola Cappello

Associate Professor

University of Turin

ITALY

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Jae-Hoon Kim

Yonsei University College of Medicine

SOUTH KOREA

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Kay-Pong Yip

Professor

University of South Florida

UNITED STATES

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Deepak Nagrath

Associate Professor

University of Michigan

UNITED STATES

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Han Cun

Assistant Professor

University of Chicago

UNITED STATES

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Jared Burks

Professor

The University of Texas MD Anderson Cancer Center

UNITED STATES

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Kwong-Kwok Wong

Professor

The University of Texas MD Anderson Cancer Center

UNITED STATES

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Samuel Mok

Research Interests

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About

Articles (11)

Molecular, Metabolic, and Subcellular Mapping of the Tumor Immune Microenvironment via 3D Targeted and Non-Targeted Multiplex Multi-Omics Analyses

Spatial Transcriptomics Depict Ligand–Receptor Cross-talk Heterogeneity at the Tumor-Stroma Interface in Long-Term Ovarian Cancer Survivors

Ryanodine receptor 1-mediated Ca<sup>2+</sup> signaling and mitochondrial reprogramming modulate uterine serous cancer malignant phenotypes.

Quality of Life and Adverse Events: Prognostic Relationships in Long-Term Ovarian Cancer Survival.

SIO: A Spatioimageomics Pipeline to Identify Prognostic Biomarkers Associated with the Ovarian Tumor Microenvironment

Dissection of PIK3CA Aberration for Cervical Adenocarcinoma Outcomes.

Correction: Hu, W., et al. Targeting Dopamine Receptor D2 by Imipridone Suppresses Uterine Serous Cancer Malignant Phenotype. Cancers 2020, 12, 2436.

ITLN1 modulates invasive potential and metabolic reprogramming of ovarian cancer cells in omental microenvironment.

Targeting Dopamine Receptor D2 by Imipridone Suppresses Uterine Serous Cancer Malignant Phenotype

Disabled-2: a positive regulator of the early differentiation of myoblasts.

Expression of long noncoding RNAs in cancer-associated fibroblasts linked to patient survival in ovarian cancer.

Collaborators (8)

Austin Miller

Paola Cappello

Jae-Hoon Kim

Kay-Pong Yip

Deepak Nagrath

Han Cun

Jared Burks

Kwong-Kwok Wong

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