Sandip M. Swain

Assistant Professor in Medicine

Duke University

United States

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Dr. Sandip M. Swain is an Assistant Professor in Medicine at Duke University, USA. His research primarily focuses on the roles of phosphate in mitochondrial function and pancreatitis, as well as the mechanisms involved in α-synuclein transfer and mechanosensing in gastrointestinal disorders. He has contributed significantly to understanding pathological changes related to pancreatic fibrosis and endothelial cell responses to shear stress.

Articles (14)

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2025

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2023

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2023

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2022

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2022

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2022

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2021

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2021

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2020

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2020

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2020

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2020

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2020

Collaborators (5)

Nirakar Sahoo

Assistant Professor

The University of Texas Rio Grande Valley

UNITED STATES

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Enquan Xu

Duke University

UNITED STATES

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Andrew West

Professor

Duke University

UNITED STATES

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Diana Imhof

W3 Professor for Pharmaceutical Biochemistry and Bioanalytics

Rheinische Friedrich-Wilhelms-Universität Bonn

GERMANY

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Toni Kühl

University of Bonn

GERMANY

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Sandip M. Swain

Research Interests

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About

Articles (14)

Phosphate Improves Mitochondrial Function and Reduces Pancreatitis in Hypertriglyceridemia

Gut mucosal cells transfer α-synuclein to the vagus nerve

Mechanosensing Piezo channels in gastrointestinal disorders

Intracellular hemin is a potent inhibitor of the voltage-gated potassium channel Kv10.1

Piezo1-mediated stellate cell activation causes pressure-induced pancreatic fibrosis in mice

Initiation and severity of experimental pancreatitis are modified by phosphate.

The Role of Phosphate in Alcohol-Induced Experimental Pancreatitis.

Heterogeneity in α-synuclein fibril activity correlates to disease phenotypes in Lewy body dementia.

Piezo1 acts upstream of TRPV4 to induce pathological changes in endothelial cells due to shear stress

Pressure‐sensing Piezo1: the eyes have it

Piezo1 acts upstream of TRPV4 to induce pathological changes in endothelial cells due to shear stress

TRPV4 channel opening mediates pressure-induced pancreatitis initiated by Piezo1 activation

TRPV4 channel opening mediates pressure-induced pancreatitis initiated by Piezo1 activation

Impact of intracellular hemin on N-type inactivation of voltage-gated K<sup>+</sup> channels

Collaborators (5)

Nirakar Sahoo

Enquan Xu

Andrew West

Diana Imhof

Toni Kühl

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